Further evidence that stress hyperthermia is a fever
dc.contributor.author | Kluger, Matthew J. | en_US |
dc.contributor.author | O'Reilly, Barbara | en_US |
dc.contributor.author | Shope, Timothy R. | en_US |
dc.contributor.author | Vander, Arthur J. | en_US |
dc.date.accessioned | 2006-04-07T20:04:04Z | |
dc.date.available | 2006-04-07T20:04:04Z | |
dc.date.issued | 1987 | en_US |
dc.identifier.citation | Kluger, Matthew J., O'Reilly, Barbara, Shope, Timothy R., Vander, Arthur J. (1987)."Further evidence that stress hyperthermia is a fever." Physiology & Behavior 39(6): 763-766. <http://hdl.handle.net/2027.42/26990> | en_US |
dc.identifier.uri | http://www.sciencedirect.com/science/article/B6T0P-482RG3N-B5/2/578b19911c9880bfe4698f4488057c32 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/26990 | |
dc.identifier.uri | http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=3602130&dopt=citation | en_US |
dc.description.abstract | Exposure of rats to an open-field results in a rapid rise in body temperature. Fifty-four percent of this rise in body temperature was blocked by intracerebroventricular administration of the antipyretic drug sodium salicylate. Intraperitoneal administration of indomethacin, a potent blocker of prostaglandin production, also attenuated the stress-induced hyperthermia to the same degree. Based on the data presented in this and an earlier study, we conclude that a major component of the rise in body temperature induced by psychological stress in rats is mediated by prostaglandins released by the central nervous system, and may therefore be a fever. | en_US |
dc.format.extent | 355015 bytes | |
dc.format.extent | 3118 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.language.iso | en_US | |
dc.publisher | Elsevier | en_US |
dc.title | Further evidence that stress hyperthermia is a fever | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Public Health | en_US |
dc.subject.hlbsecondlevel | Psychology | en_US |
dc.subject.hlbsecondlevel | Neurosciences | en_US |
dc.subject.hlbsecondlevel | Molecular, Cellular and Developmental Biology | en_US |
dc.subject.hlbtoplevel | Social Sciences | en_US |
dc.subject.hlbtoplevel | Science | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | Department of Physiology, The University of Michigan Medical School, Ann Arbor, MI 48109, USA | en_US |
dc.contributor.affiliationum | Department of Physiology, The University of Michigan Medical School, Ann Arbor, MI 48109, USA | en_US |
dc.contributor.affiliationum | Department of Physiology, The University of Michigan Medical School, Ann Arbor, MI 48109, USA | en_US |
dc.contributor.affiliationum | Department of Physiology, The University of Michigan Medical School, Ann Arbor, MI 48109, USA | en_US |
dc.identifier.pmid | 3602130 | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/26990/1/0000557.pdf | en_US |
dc.identifier.doi | http://dx.doi.org/10.1016/0031-9384(87)90263-0 | en_US |
dc.identifier.source | Physiology & Behavior | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
Files in this item
Remediation of Harmful Language
The University of Michigan Library aims to describe library materials in a way that respects the people and communities who create, use, and are represented in our collections. Report harmful or offensive language in catalog records, finding aids, or elsewhere in our collections anonymously through our metadata feedback form. More information at Remediation of Harmful Language.
Accessibility
If you are unable to use this file in its current format, please select the Contact Us link and we can modify it to make it more accessible to you.