Emergent percutaneous cardiopulmonary bypass in patients having cardiovascular collapse in the cardiac catheterization laboratory
dc.contributor.author | Grambow, David W. | en_US |
dc.contributor.author | Michael Deeb, G. | en_US |
dc.contributor.author | Pavlides, Gregory S. | en_US |
dc.contributor.author | Margulis, Ann | en_US |
dc.contributor.author | O'Neill, William W. | en_US |
dc.contributor.author | Bates, Eric R. | en_US |
dc.date.accessioned | 2006-04-10T18:11:58Z | |
dc.date.available | 2006-04-10T18:11:58Z | |
dc.date.issued | 1994-05-01 | en_US |
dc.identifier.citation | Grambow, David W., Michael Deeb, G., Pavlides, Gregory S., Margulis, Ann, O'Neill, William W., Bates, Eric R. (1994/05/01)."Emergent percutaneous cardiopulmonary bypass in patients having cardiovascular collapse in the cardiac catheterization laboratory." The American Journal of Cardiology 73(12): 872-875. <http://hdl.handle.net/2027.42/31621> | en_US |
dc.identifier.uri | http://www.sciencedirect.com/science/article/B6T10-4C7V5WM-HK/2/3a21c8c6d3d28502a73b06c09234d646 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/31621 | |
dc.identifier.uri | http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=8184811&dopt=citation | en_US |
dc.description.abstract | Percutaneous cardiopulmonary bypass (PCB) was instituted in 30 initially stable patients who developed either cardiac arrest refractory to resuscitation (n = 7) or cardiogenic shock (mean arterial blood pressure <50 mm Hg unresponsive to fluid resuscitation or vasopressors) (n = 23) after a cathetertzation laboratory complication. Events leading to collapse included abrupt closure during percutaneous transluminal coronary angioplasty (PTCA) (n = 22), complications from diagnostic cardiac catheterization (n = 6), left ventricular perforation during mural valvuloplasty (n = 1), and right ventricular perforation during pericardiocentesis (n = 1). PCB was initiated within 20 minutes of cardiovascular collapse in 83% of patients (arrest: 21 +/- 13 minutes [range 10 to 50]; and shock: 17 +/- 6 minutes [range 10 to 30]). Mean arterial blood pressure increased on PCB from 0 to 56 mm Hg in patients with cardiac arrest and from 37 to 63 mm Hg in those with cardiogenic shock at mean PCB flow rates of 2.5 to 5.0 liters/min. Subsequent therapy on PCB included emergent cardiac surgery (n = 14), PTCA (n = 13) and medical therapy (n = 3). Six patients (20%) survived to hospital discharge (3 with cardiac surgery, 2 with PTCA, and 1 with medical therapy). All 7 patients with refractory cardiac arrest died despite further interventions on PCB, whereas 6 of 23 (26%) with cardiogenic shock survived to hospital discharge. Thus, in response to cardiovascular collapse in the catheterization laboratory, PCB does not salvage patients who do not regain a stable cardiac rhythm. PCB can stabilize patients who develop cardiogenic shock for further interventions which are lifesaving in only a minority of patients. | en_US |
dc.format.extent | 481244 bytes | |
dc.format.extent | 3118 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.language.iso | en_US | |
dc.publisher | Elsevier | en_US |
dc.title | Emergent percutaneous cardiopulmonary bypass in patients having cardiovascular collapse in the cardiac catheterization laboratory | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Internal Medicine and Specialties | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA; Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA; Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA; Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA; Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA; Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Cardiology Division, Department of Internal Medicine, and Department of Thoracic Surgery, University of Michigan, Ann Arbor, Michigan, USA; Department of Cardiology, William Beaumont Hospital, Royal Oak, Michigan, USA. | en_US |
dc.identifier.pmid | 8184811 | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/31621/1/0000554.pdf | en_US |
dc.identifier.doi | http://dx.doi.org/10.1016/0002-9149(94)90813-3 | en_US |
dc.identifier.source | The American Journal of Cardiology | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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