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Genomic copy number changes of DNA repair genes ERCC1 and ERCC2 in human gliomas

dc.contributor.authorReed, Eddieen_US
dc.contributor.authorLiang, Bertrand C.en_US
dc.contributor.authorRoss, Donald A.en_US
dc.date.accessioned2006-09-11T15:59:48Z
dc.date.available2006-09-11T15:59:48Z
dc.date.issued1995-10en_US
dc.identifier.citationLiang, Bertrand C.; Ross, Donald A.; Reed, Eddie; (1995). "Genomic copy number changes of DNA repair genes ERCC1 and ERCC2 in human gliomas." Journal of Neuro-Oncology 26(1): 17-23. <http://hdl.handle.net/2027.42/45384>en_US
dc.identifier.issn0167-594Xen_US
dc.identifier.issn1573-7373en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/45384
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=8583241&dopt=citationen_US
dc.description.abstractAbnormalities of the genomic region of chromosome 19q13.2–13.4 are a common occurrence in brain malignancies and contain a possible tumor suppressor gene involved in gliomas. Since abnormalities of DNA repair are associated with malignancy, we assessed DNA status of the nucleotide excision repair genes located in this area, viz. ERCC1 and ERCC2.en_US
dc.format.extent840643 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherKluwer Academic Publishers; Springer Science+Business Mediaen_US
dc.subject.otherERCC1en_US
dc.subject.otherDNA Repairen_US
dc.subject.otherMedicine & Public Healthen_US
dc.subject.otherOncologyen_US
dc.subject.otherERCC2en_US
dc.subject.otherGliomaen_US
dc.titleGenomic copy number changes of DNA repair genes ERCC1 and ERCC2 in human gliomasen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPublic Healthen_US
dc.subject.hlbsecondlevelOtolaryngologyen_US
dc.subject.hlbsecondlevelOphthalmologyen_US
dc.subject.hlbsecondlevelOncology and Hematologyen_US
dc.subject.hlbsecondlevelObstetrics and Gynecologyen_US
dc.subject.hlbsecondlevelNeurosciencesen_US
dc.subject.hlbsecondlevelInternal Medicine and Specialtiesen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumSection of Neurosurgery, Department of Surgery, University of Michigan Medical School, Ann Arbor, MI, USAen_US
dc.contributor.affiliationotherClinical Pharmacology Branch, Division of Cancer Treatment, National Cancer Institute, National Institutes of Health, Bethesda, MD; UCHSC, 4200 E. Ninth Ave., Box B182, 80262, Denver, CO, USAen_US
dc.contributor.affiliationotherClinical Pharmacology Branch, Division of Cancer Treatment, National Cancer Institute, National Institutes of Health, Bethesda, MDen_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid8583241en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/45384/1/11060_2005_Article_BF01054765.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/BF01054765en_US
dc.identifier.sourceJournal of Neuro-Oncologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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