View Item 

Show simple item record

Protein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Association

dc.contributor.authorNistor, Ioanaen_US
dc.contributor.authorNair, Rajan P.en_US
dc.contributor.authorStuart, Philipen_US
dc.contributor.authorHiremagalore, Ravien_US
dc.contributor.authorThompson, Rachel A.en_US
dc.contributor.authorJenisch, Stefanen_US
dc.contributor.authorWeichenthal, Michaelen_US
dc.contributor.authorAbecasis, Gonçalo R.en_US
dc.contributor.authorQin, Zhaohui Steveen_US
dc.contributor.authorChristophers, Ennoen_US
dc.contributor.authorLim, Henry W.en_US
dc.contributor.authorVoorhees, John J.en_US
dc.contributor.authorElder, James T.en_US
dc.date.accessioned2010-06-01T21:21:43Z
dc.date.available2010-06-01T21:21:43Z
dc.date.issued2005-05en_US
dc.identifier.citationNistor, Ioana; Nair, Rajan P.; Stuart, Philip; Hiremagalore, Ravi; Thompson, Rachel A.; Jenisch, Stefan; Weichenthal, Michael; Abecasis, GonÇalo R.; Qin, Zhaohui S.; Christophers, Enno; Lim, Henry W.; Voorhees, John J.; Elder, James T. (2005). "Protein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Association." Journal of General Internal Medicine 20(5): 395-396. <http://hdl.handle.net/2027.42/74426>en_US
dc.identifier.issn0884-8734en_US
dc.identifier.issn1525-1497en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/74426
dc.format.extent57066 bytes
dc.format.extent3109 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherBlackwell Science Incen_US
dc.rightsCopyright © 2005 by The Society for Investigative Dermatology, Inc.en_US
dc.titleProtein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Associationen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelInternal Medicine and Specialtiesen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Dermatology, University of Michigan Medical School, Ann Arbor, Michigan, USA ; Departments ofen_US
dc.contributor.affiliationum§ Department of Biostatistics, Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, Michigan, USA ;en_US
dc.contributor.affiliationum¶ Department of Dermatology, Henry Ford Hospital, Detroit, Michigan, USA ;en_US
dc.contributor.affiliationum∥ Department of Radiation Oncology (Cancer Biology), University of Michigan Medical School, Ann Arbor, Michigan, USA ;en_US
dc.contributor.affiliationum# Ann Arbor Veterans Affairs Hospital, Ann Arbor, Michigan, USAen_US
dc.contributor.affiliationother† Immunology anden_US
dc.contributor.affiliationother† Dermatology, University of Kiel, Kiel, Germany ;en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/74426/1/j.0022-202X.2005.23802.x.pdf
dc.identifier.doi10.1111/j.0022-202X.2005.23802.xen_US
dc.identifier.sourceJournal of General Internal Medicineen_US
dc.identifier.citedreferenceBegovich AB, Caillier SJ, Alexander HC, Penko JM, Hauser SL, Barcellos LF, Oksenberg JR: The R620W polymorphism of the protein tyrosine phosphatase PTPN22 is not associated with multiple sclerosis. Am J Hum Genet 76: 184 – 187, 2005en_US
dc.identifier.citedreferenceBegovich AB, Carlton VE, Honigberg LA, Schrodi SJ, Chokkalingam AP, Alexander HC, Ardlie KG, et al: A missense single-nucleotide polymorphism in a gene encoding a protein tyrosine phosphatase (PTPN22) is associated with rheumatoid arthritis. Am J Hum Genet 75: 330 – 337, 2004en_US
dc.identifier.citedreferenceBottini N, Musumeci L, Alonso A, et al: A functional variant of lymphoid tyrosine phosphatase is associated with type I diabetes. Nat Genet 36: 337 – 338, 2004en_US
dc.identifier.citedreferenceCloutier JF, Veillette A: Association of inhibitory tyrosine protein kinase p50csk with protein tyrosine phosphatase PEP in T cells and other hemopoietic cells. EMBO J 15: 4909 – 4918, 1996en_US
dc.identifier.citedreferenceCloutier JF, Veillette A: Cooperative inhibition of T-cell antigen receptor signaling by a complex between a kinase and a phosphatase. J Exp Med 189: 111 – 121, 1999en_US
dc.identifier.citedreferenceCollaboration: Fine mapping of the psoriasis susceptibiliity gene PSORS1: A reassessment of risk associated with a putative risk haplotype lacking HLA-Cw6. J Invest Dermatol, 2005, in pressen_US
dc.identifier.citedreferenceCriswell LA, Pfeiffer KA, Lum RF, et al: Analysis of families in the Multiple Autoimmune Disease Genetics Consortium (MADGC) collection: The PTPN22 620W allele associates with multiple autoimmune phenotypes. Am J Hum Genet 76: 561 – 571, 2005en_US
dc.identifier.citedreferenceHorvath S, Xu X, Laird NM: The family based association test method: Strategies for studying general genotype–phenotype associations. Eur J Hum Genet 9: 301 – 306, 2001en_US
dc.identifier.citedreferenceHorvath S, Xu X, Lake SL, Silverman EK, Weiss ST, Laird NM: Family-based tests for associating haplotypes with general phenotype data: Application to asthma genetics. Genet Epidemiol 26: 61 – 69, 2004en_US
dc.identifier.citedreferenceKnapp M: A note on power approximations for the transmission/disequilibrium test. Am J Hum Genet 64: 1177 – 1185, 1999en_US
dc.identifier.citedreferenceKyogoku C, Langefeld CD, Ortmann WA, et al: Genetic association of the R620W polymorphism of protein tyrosine phosphatase PTPN22 with human SLE. Am J Hum Genet 75: 504 – 507, 2004en_US
dc.identifier.citedreferenceLadner MB, Bottini N, Valdes AM, Noble JA: Association of the single nucleotide polymorphism C1858T of the PTPN22 gene with type 1 diabetes. Hum Immunol 66: 60 – 64, 2005en_US
dc.identifier.citedreferenceLew W, Bowcock AM, Krueger JG: Psoriasis vulgaris: Cutaneous lymphoid tissue supports T-cell activation and “Type 1” inflammatory gene expression. Trends Immunol 25: 295 – 305, 2004en_US
dc.identifier.citedreferenceMartin ER, Bass MP, Kaplan NL: Correcting for a potential bias in the pedigree disequilibrium test. Am J Hum Genet 68: 1065 – 1067, 2001en_US
dc.identifier.citedreferenceMartin ER, Monks SA, Warren LL, Kaplan NL: A test for linkage and association in general pedigrees: The pedigree disequilibrium test. Am J Hum Genet 67: 146 – 154, 2000en_US
dc.identifier.citedreferenceOnengut-Gumuscu S, Ewens KG, Spielman RS, Concannon P: A functional polymorphism (1858C/T) in the PTPN22 gene is linked and associated with type I diabetes in multiplex families. Genes Immun 5: 678 – 680, 2004en_US
dc.identifier.citedreferenceRabinowitz D, Laird N: A unified approach to adjusting association tests for population admixture with arbitrary pedigree structure and arbitrary missing marker information. Hum Heredity 50: 211 – 223, 2000en_US
dc.identifier.citedreferenceSmyth D, Cooper JD, Collins JE, et al: Replication of an association between the lymphoid tyrosine phosphatase locus (LYP/PTPN22) with type 1 diabetes, and evidence for its role as a general autoimmunity locus. Diabetes 53: 3020 – 3023, 2004en_US
dc.identifier.citedreferenceSpielman RS, Ewens WJ: The TDT and other family-based tests for linkage disequilibrium and association. Am J Hum Genet 59: 983 – 989, 1996en_US
dc.identifier.citedreferenceSpielman RS, McGinnis RE, Ewens WJ: Transmission test for linkage disequilibrium: The insulin gene region and insulin-dependent diabetes mellitus (IDDM). Am J Hum Genet 52: 506 – 516, 1993en_US
dc.identifier.citedreferenceSugiyama H, Gyulai R, Toichi E, et al: Dysfunctional blood and target tissue CD4+CD25 high regulatory T cells in psoriasis: Mechanism underlying unrestrained pathogenic effector T cell proliferation. J Immunol 174: 164 – 173, 2005en_US
dc.identifier.citedreferenceVelaga MR, Wilson V, Jennings CE, et al: The codon 620 tryptophan allele of the lymphoid tyrosine phosphatase (LYP) gene is a major determinant of Graves' disease. J Clin Endocrinol Metab 89: 5862 – 5865, 2004en_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


Files in this item

Name:
j.0022-202X.2005.23802.x.pdf
Size:
55.72KB
Format:
PDF
View/Open

Show simple item record

Remediation of Harmful Language

The University of Michigan Library aims to describe library materials in a way that respects the people and communities who create, use, and are represented in our collections. Report harmful or offensive language in catalog records, finding aids, or elsewhere in our collections anonymously through our metadata feedback form. More information at Remediation of Harmful Language.

Accessibility

If you are unable to use this file in its current format, please select the Contact Us link and we can modify it to make it more accessible to you.