View Item 

Show simple item record

Inactivation of Kell blood group antigens by 2-aminoethylisothiouronium bromide

dc.contributor.authorAdvani, H.en_US
dc.contributor.authorZamor, J.en_US
dc.contributor.authorJudd, W. Johnen_US
dc.contributor.authorJohnson, C. L.en_US
dc.contributor.authorMarsh, W. L.en_US
dc.date.accessioned2010-06-01T21:58:36Z
dc.date.available2010-06-01T21:58:36Z
dc.date.issued1982-05en_US
dc.identifier.citationAdvani, H.; Zamor, J.; Judd, W. J.; Johnson, C. L.; Marsh, W. L. (1982). "Inactivation of Kell blood group antigens by 2-aminoethylisothiouronium bromide." British Journal of Haematology 51(1): 107-115. <http://hdl.handle.net/2027.42/75007>en_US
dc.identifier.issn0007-1048en_US
dc.identifier.issn1365-2141en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/75007
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=7073987&dopt=citationen_US
dc.format.extent538017 bytes
dc.format.extent3109 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherBlackwell Publishing Ltden_US
dc.rights1982 Blackwell Publishing Ltden_US
dc.titleInactivation of Kell blood group antigens by 2-aminoethylisothiouronium bromideen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelOncology and Hematologyen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumThe Lindsley F. Kimball Research Institute of The New York Blood Center, New York, and Department of Pathology, University of Michigan, Ann Arboren_US
dc.identifier.pmid7073987en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/75007/1/j.1365-2141.1982.tb07295.x.pdf
dc.identifier.doi10.1111/j.1365-2141.1982.tb07295.xen_US
dc.identifier.sourceBritish Journal of Haematologyen_US
dc.identifier.citedreferenceALLEN, F.H., KRABBE, S.M.R. & CORCORAN, P.A. ( 1961 ) A new phenotype (McLeod) in the Kell blood group system. Vox Sanguinis, 6, 555 – 560.en_US
dc.identifier.citedreferenceBECK, M.L., MARSH, W.L., PIERCE, S.R., DI NAPOLI, J., ØYEN, R. & NICHOLS, M.E. ( 1979 ) Auto-anti-Kp b associated with weakened antigenicity in the Kell blood-group system: a second example. Transfusion, 19, 197 – 202.en_US
dc.identifier.citedreferenceBURAPAKULSOLSRI, N., YUTHAVONG, Y. & WILAIRAT, P. ( 1979 ) An examination of complement proteins on membranes of paroxysmal nocturnal haemoglobinuria (PNH) and PNH-like red cells. British Journal of Haematology, 41, 393 – 398.en_US
dc.identifier.citedreferenceCANELLOS, G.P., BRAIN, M.C. & DACIE, J.V. ( 1970 ) Effects of sulphydryl inhibition on the erythrocyte in paroxysmal nocturnal haemoglobinuria. British Journal of Haematology, 18, 269 – 277.en_US
dc.identifier.citedreferenceCHOWN, B., LEWIS, M. & KAITA, H. ( 1957 ) A “new” Kell blood group phenotype. Nature, 180, 711.en_US
dc.identifier.citedreferenceDE SANDRE, G., CORTESI, S., CORROCHER, R., FALEZZA, G. & PERONA, G. ( 1970 ) On the mechanism of the in vitro production of Ham-positive red cells by sulphydryl compounds: role of pH and free -SH groups. British Journal of Haematology, 18, 551 – 556.en_US
dc.identifier.citedreferenceFERRONE, S., MARUBINI, E., MERCURIALI, F. & SIRCHIA, G. ( 1972 ) Study of in vitro lysis of paroxysmal nocturnal haemoglobinuria (PNH) and PNH-like red cells. British Journal of Haematology, 23, 5 – 12.en_US
dc.identifier.citedreferenceGIBLETT, E.R. ( 1961 ) A critique of the theoretical hazard of inter- vs. intraracial transfusion. Transfusion, 1, 233 – 238.en_US
dc.identifier.citedreferenceISSITT, P.D. & ISSITT, C.H. ( 1975 ) Applied Blood Group Serology, 2nd edn, p. 167. Spectra Bio-logicals, Oxnard, California.en_US
dc.identifier.citedreferenceKORNSTAD, L. & HEISTÖ, H. ( 1957 ) The frequency of formation of Kell antibodies in recipients of Kell positive blood. Proceedings of the 6th Congress of European Society of Haematology, p. 754.en_US
dc.identifier.citedreferenceLEWIS, S.M., LAMBERTENGHI, G., FERRONE, S. & SIRCHIA, G. ( 1971 ) Electron microscope study of PNH red cells and AET-treated normal red cells (PNH-like cells). Journal of Clinical Pathology, 24, 677 – 684.en_US
dc.identifier.citedreferenceMARSH, W.L. ( 1972 ) Scoring of hemagglutination reactions. Transfusion, 12, 352 – 353.en_US
dc.identifier.citedreferenceMARSH, W.L. ( 1975 ) Studies on the Kell blood group system. Medical Laboratory Technology, 32, 1 – 18.en_US
dc.identifier.citedreferenceMARSH, W.L. ( 1978 ) Chronic granulomatous disease, the McLeod syndrome, and the Kell blood groups. Birth Defects: Original Article Series (The National Foundation), 14, 9 – 25.en_US
dc.identifier.citedreferenceMARSH, W.L., TASWELL, H.F., ØYEN, R., NICHOLS, M.E., VERGARA, M.S. & PINEDA, A.A. ( 1975a ) Kx antigen of the Kell system and its relationship to chronic granulomatous disease. Evidence that the Kx gene is X-linked. Transfusion, 15, 527.en_US
dc.identifier.citedreferenceMARSH, W.L., ØYEN, R., NICHOLS, M.E. & ALLEN, F.H., JR ( 1975b ) Chronic granulomatous disease and the Kell blood groups. British Journal of Haematology, 29, 247 – 262.en_US
dc.identifier.citedreferenceMARSH, W.L., DI NAPOLI, J. & ØYEN, R. ( 1979a ) Auto-immune hemolytic anemia caused by anti-K13. Vox Sanguinis, 36, 174 – 178.en_US
dc.identifier.citedreferenceMARSH, W.L., ØYEN, R., ALICEA, E., LINTER, M. & HORTON, S. ( 1979b ) Auto-immune hemolytic anemia and the Kell blood groups. American Journal of Hematology, 7, 155 – 162.en_US
dc.identifier.citedreferenceMULLER, A., ANDRÉ-LIARDET, J., GARRETTA, M., BROCTEUR, J. & MOULLEC, J. ( 1973 ) Observations sur un anticorps rare: l'anti-Gerbich. Revue Francais de Transfusion, 16, 251 – 255.en_US
dc.identifier.citedreferenceRUBIN, H. ( 1963 ) Antibody elution from red blood cells. Journal of Clinical Pathology, 16, 70 – 73.en_US
dc.identifier.citedreferenceSABO, B., MCCREARY, J., STROUP, M., SMITH, D.E. & WEIDNER, J.G. ( 1979 ) Another Kell-related antibody, anti-K19. Vox Sanguinis, 36, 97 – 102.en_US
dc.identifier.citedreferenceSEYFRIED, H., GORSKA, B., MAJ, S., SYLWESTROWICZ, T., GILES, C.M. & GOLDSMITH, K.L.G. ( 1972 ) Apparent depression of antigens of the Kell blood group system associated with autoimmune acquired hemolytic anemia. Vox Sanguinis, 23, 528 – 536.en_US
dc.identifier.citedreferenceSIRCHIA, G., FERRONE, S. & MERCURIALI, F. ( 1965 ) The action of two sulphydryl compounds on normal human red cells. Relationship to red cells of paroxysmal nocturnal hemoglobinuria. Blood, 25, 502 – 510.en_US
dc.identifier.citedreferenceSIRCHIA, G., FERRONE, S., MILANI, R. & MERCURIALI, F. ( 1966 ) Observations on certain enzyme activities of normal human red cells treated with sulphydryl compounds. Blood, 28, 98 – 101.en_US
dc.identifier.citedreferenceSIRCHIA, G. & DACIE, J.V. ( 1967 ) Immune lysis of AET-treated normal red cells (PNH-like cells). Nature, 215, 747 – 748.en_US
dc.identifier.citedreferenceSIRCHIA, G. & FERRONE, S. ( 1972 ) The laboratory substitutes of the red cell of paroxysmal nocturnal haemoglobinuria (PNH): PNH-like red cells. Series Haematologica, 5, ( 3 ), 137 – 175.en_US
dc.identifier.citedreferenceSYMMANS, W.A., SHEPHERD, C.S., MARSH, W.L., ØYEN, R., SHOHET, S.B. & LINEHAN, B.J. ( 1979 ) Hereditary acanthocytosis associated with the McLeod phenotype of the Kell blood group system. British Journal of Haematology, 42, 575 – 583.en_US
dc.identifier.citedreferenceWHITE, W., WASHINGTON, E.D., SABO, B.H., STROUP, M., MCCREARY, J., ØYEN, R. & MARSH, W.L. ( 1980 ) Anti-Km in a transfused man with McLeod syndrome. Blood Transfusion and Immunohaematology, 23, 305 – 317.en_US
dc.identifier.citedreferenceWIMBER, B.M., MARSH, W.L., TASWELL, H.F. & GALEY, W.R. ( 1977 ) Haematological changes associated with the McLeod phenotype of the Kell blood group system. British Journal of Haematology, 36, 219 – 224.en_US
dc.identifier.citedreferenceYAMAGUCHI, H., OKUBO, Y., SENO, T., MATSUSHITA, K. & DANIELS, G.L. ( 1979 ) A “new” allele, Kp c, at the Kell complex locus. Vox Sanguinis, 36, 29 – 33.en_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


Files in this item

Name:
j.1365-2141.1982.tb07295.x.pdf
Size:
525.4KB
Format:
PDF
View/Open

Show simple item record

Remediation of Harmful Language

The University of Michigan Library aims to describe library materials in a way that respects the people and communities who create, use, and are represented in our collections. Report harmful or offensive language in catalog records, finding aids, or elsewhere in our collections anonymously through our metadata feedback form. More information at Remediation of Harmful Language.

Accessibility

If you are unable to use this file in its current format, please select the Contact Us link and we can modify it to make it more accessible to you.