Cyclin D 1‐induced proliferation is independent of beta‐catenin in H ead and N eck C ancer
dc.contributor.author | Sales, KU | en_US |
dc.contributor.author | Giudice, FS | en_US |
dc.contributor.author | Castilho, RM | en_US |
dc.contributor.author | Salles, FT | en_US |
dc.contributor.author | Squarize, CH | en_US |
dc.contributor.author | Abrahao, AC | en_US |
dc.contributor.author | Pinto, DS | en_US |
dc.date.accessioned | 2014-05-21T18:03:33Z | |
dc.date.available | WITHHELD_13_MONTHS | en_US |
dc.date.available | 2014-05-21T18:03:33Z | |
dc.date.issued | 2014-04 | en_US |
dc.identifier.citation | Sales, KU; Giudice, FS; Castilho, RM; Salles, FT; Squarize, CH; Abrahao, AC; Pinto, DS (2014). "Cyclin D 1‐induced proliferation is independent of beta‐catenin in H ead and N eck C ancer." Oral Diseases 20(3): e42-e48. | en_US |
dc.identifier.issn | 1354-523X | en_US |
dc.identifier.issn | 1601-0825 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/106775 | |
dc.publisher | Wiley Periodicals, Inc. | en_US |
dc.subject.other | Head and Neck Squamous Cell Carcinoma | en_US |
dc.subject.other | Beta‐Catenin | en_US |
dc.subject.other | A Kt | en_US |
dc.subject.other | Cyclin D 1 | en_US |
dc.title | Cyclin D 1‐induced proliferation is independent of beta‐catenin in H ead and N eck C ancer | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Dentistry | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/106775/1/odi12124.pdf | |
dc.identifier.doi | 10.1111/odi.12124 | en_US |
dc.identifier.source | Oral Diseases | en_US |
dc.identifier.citedreference | Molinolo AA, Amornphimoltham P, Squarize CH, Castilho RM, Patel V, Gutkind JS ( 2009 ). Dysregulated molecular networks in head and neck carcinogenesis. Oral Oncol 45: 324 – 334. | en_US |
dc.identifier.citedreference | Diehl JA, Zindy F, Sherr CJ ( 1997 ). Inhibition of cyclin D1 phosphorylation on threonine‐286 prevents its rapid degradation via the ubiquitin‐proteasome pathway. Genes Dev 11: 957 – 972. | en_US |
dc.identifier.citedreference | Diehl JA, Cheng M, Roussel MF, Sherr CJ ( 1998 ). Glycogen synthase kinase‐3beta regulates cyclin D1 proteolysis and subcellular localization. Genes Dev 12: 3499 – 3511. | en_US |
dc.identifier.citedreference | Döbrossy L ( 2005 ). Epidemiology of head and neck cancer: magnitude of the problem. Cancer Metastasis Rev 24: 9 – 17. | en_US |
dc.identifier.citedreference | Filippa N, Sable CL, Hemmings BA, Van Obberghen E ( 2000 ). Effect of phosphoinositide‐dependent kinase 1 on protein kinase B translocation and its subsequent activation. Mol Cell Biol 20: 5712 – 5721. | en_US |
dc.identifier.citedreference | Franke TF, Yang SI, Chan TO et al ( 1995 ). The protein kinase encoded by the Akt proto‐oncogene is a target of the PDGF‐activated phosphatidylinositol 3‐kinase. Cell 81: 727 – 736. | en_US |
dc.identifier.citedreference | Hoschuetzky H, Aberle H, Kemler R ( 1994 ). Beta‐catenin mediates the interaction of the cadherin‐catenin complex with epidermal growth factor receptor. J Cell Biol 127: 1375 – 1380. | en_US |
dc.identifier.citedreference | Iwai S, Yonekawa A, Harada C et al ( 2010 ). Involvement of the Wnt‐β‐catenin pathway in invasion and migration of oral squamous carcinoma cells. Int J Oncol 37: 1095 – 1103. | en_US |
dc.identifier.citedreference | Jamora C, Fuchs E ( 2002 ). Intercellular adhesion, signalling and the cytoskeleton. Nat Cell Biol 4: E101 – E108. | en_US |
dc.identifier.citedreference | Ji H, Wang J, Nika H et al ( 2009 ). EGF‐induced ERK activation promotes CK2‐mediated disassociation of alpha‐Catenin from beta‐Catenin and transactivation of beta‐Catenin. Mol Cell 36: 547 – 559. | en_US |
dc.identifier.citedreference | Kam Y, Quaranta V ( 2009 ). Cadherin‐bound beta‐catenin feeds into the Wnt pathway upon adherents junctions dissociation: evidence for an intersection between beta‐catenin pools. PLoS One 4: e4580. | en_US |
dc.identifier.citedreference | Liu F, Millar SE ( 2010 ). Wnt/beta‐catenin signaling in oral tissue development and disease. J Dent Res 89: 318 – 330. | en_US |
dc.identifier.citedreference | Liu C, Tu Y, Sun X et al ( 2011 ). Wnt/beta‐Catenin pathway in human glioma: expression pattern and clinical/prognostic correlations. Clin Exp Med 11: 105 – 112. | en_US |
dc.identifier.citedreference | Lo Muzio L ( 2001 ). A possible role for the WNT‐1 pathway in oral carcinogenesis. Crit Rev Oral Biol Med 12: 152 – 165. | en_US |
dc.identifier.citedreference | Miyamoto R, Uzawa N, Nagaoka S, Hirata Y, Amagasa T ( 2003 ). Prognostic significance of cyclin D1 amplification and overexpression in oral squamous cell carcinomas. Oral Oncol 39: 610 – 618. | en_US |
dc.identifier.citedreference | Nishimura I, Ohishi Y, Oda Y et al ( 2011 ). Expression and localization of E‐cadherin and β‐catenin in uterine carcinosarcoma. Virchows Arch 458: 85 – 94. | en_US |
dc.identifier.citedreference | Polakis P ( 2000 ). Wnt signaling and cancer. Genes Dev 14: 1837 – 1851. | en_US |
dc.identifier.citedreference | Richter P, Umbreit C, Franz M et al ( 2011 ). EGF/TGFβ1 co‐stimulation of oral squamous cell carcinoma cells causes an epithelial‐mesenchymal transition cell phenotype expressing laminin 332. J Oral Pathol Med 40: 46 – 54. | en_US |
dc.identifier.citedreference | Segrelles C, Ruiz S, Perez P et al ( 2002 ). Functional roles of Akt signaling in mouse skin tumorigenesis. Oncogene 21: 53 – 64. | en_US |
dc.identifier.citedreference | Staal SP ( 1987 ). Molecular cloning of the akt oncogene and its human homologues AKT1 and AKT2: amplification of AKT1 in a primary human gastric adenocarcinoma. Proc Natl Acad Sci USA 84: 5034 – 5037. | en_US |
dc.identifier.citedreference | Suzuki H, Masuda N, Shimura T et al ( 2008 ). Nuclear beta‐catenin expression at the invasive front and in the vessels predicts liver metastasis in colorectal carcinoma. Anticancer Res 28: 1821 – 1830. | en_US |
dc.identifier.citedreference | Vivanco I, Sawyers CL ( 2002 ). The phosphatidylinositol 3‐Kinase AKT pathway in human cancer. Nat Rev Cancer 2: 489 – 501. | en_US |
dc.identifier.citedreference | Yu Z, Weinberger PM, Provost E et al ( 2005 ). beta‐Catenin functions mainly as an adhesion molecule in patients with squamous cell cancer of the head and neck. Clin Cancer Res 11: 2471 – 2477. | en_US |
dc.identifier.citedreference | Aamodt R, Bondi J, Andersen SN, Bakka A, Bukholm G, Bukholm IR ( 2010 ). The prognostic impact of protein expression of E‐cadherin‐catenin complexes differs between rectal and colon carcinoma. Gastroenterol Res Pract 2010: pii: 616023. | en_US |
dc.identifier.citedreference | Bagutti C, Speight PM, Watt FM ( 1998 ). Comparison of integrin, cadherin, and catenin expression in squamous cell carcinomas of the oral cavity. J Pathol 186: 8 – 16. | en_US |
dc.identifier.citedreference | Cardinali M, Pietraszkiewicz H, Ensley JF, Robbins KC ( 1995 ). Tyrosine phosphorylation as a marker for aberrantly regulated growth‐promoting pathways in cell lines derived from head and neck malignancies. Int J Cancer 61: 98 – 103. | en_US |
dc.identifier.citedreference | Cully M, You H, Levine AJ, Mak TW ( 2006 ). Beyond PTEN mutations: the PI3K pathway as an integrator of multiple inputs during tumorigenesis. Nat Rev Cancer 6: 184 – 192. | en_US |
dc.identifier.citedreference | Czerninski R, Amornphimoltham P, Patel V, Molinolo AA, Gutkind JS ( 2009 ). Targeting mammalian target of rapamycin by rapamycin prevents tumor progression in an oral‐specific chemical carcinogenesis model. Cancer Prev Res (Phila) 2: 27 – 36. | en_US |
dc.identifier.citedreference | Dajani OF, Meisdalen K, Guren TK et al ( 2008 ). Prostaglandin E2 upregulates EGF‐stimulated signaling in mitogenic pathways involving Akt and ERK in hepatocytes. J Cell Physiol 214: 371 – 380. | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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