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Golgi defects enhance APP amyloidogenic processing in Alzheimer's disease
dc.contributor.authorJoshi, Gunjanen_US
dc.contributor.authorWang, Yanzhuangen_US
dc.date.accessioned2015-03-05T18:24:47Z
dc.date.available2016-05-10T20:26:28Zen
dc.date.issued2015-03en_US
dc.identifier.citationJoshi, Gunjan; Wang, Yanzhuang (2015). "Golgi defects enhance APP amyloidogenic processing in Alzheimer's disease." BioEssays 37(3): 240-247.en_US
dc.identifier.issn0265-9247en_US
dc.identifier.issn1521-1878en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/110763
dc.publisherWiley Periodicals, Inc.en_US
dc.subject.otherAlzheimer's diseaseen_US
dc.subject.otherGolgi defectsen_US
dc.subject.otherGRASP55en_US
dc.subject.otherGRASP65en_US
dc.subject.otherneuronal functionen_US
dc.subject.otheramyloid precursor proteinen_US
dc.subject.otheramyloid betaen_US
dc.titleGolgi defects enhance APP amyloidogenic processing in Alzheimer's diseaseen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelMolecular, Cellular and Developmental Biologyen_US
dc.subject.hlbsecondlevelNatural Resources and Environmenten_US
dc.subject.hlbsecondlevelEcology and Evolutionary Biologyen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/110763/1/bies201400116.pdf
dc.identifier.doi10.1002/bies.201400116en_US
dc.identifier.sourceBioEssaysen_US
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dc.owningcollnameInterdisciplinary and Peer-Reviewed


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