An inability of [gamma]-acetylenic GABA to block eating evoked by hypothalamic stimulation
dc.contributor.author | Valenstein, Elliot S. | en_US |
dc.contributor.author | Myslobodsky, Michael S. | en_US |
dc.date.accessioned | 2006-04-07T17:29:49Z | |
dc.date.available | 2006-04-07T17:29:49Z | |
dc.date.issued | 1980 | en_US |
dc.identifier.citation | Valenstein, Elliot S., Myslobodsky, Michael S. (1980)."An inability of [gamma]-acetylenic GABA to block eating evoked by hypothalamic stimulation." Behavioural Brain Research 1(2): 343-349. <http://hdl.handle.net/2027.42/23406> | en_US |
dc.identifier.uri | http://www.sciencedirect.com/science/article/B6SYP-4FW093W-W/2/b15b771ee6081ce9f73146e514651688 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/23406 | |
dc.identifier.uri | http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=7295383&dopt=citation | en_US |
dc.description.abstract | Several lines of evidence have implicated the nigrostriatal dopamine system and the neocortex for eating occurring either spontaneously or in response to electrical stimulation of the lateral hypothalamus (ESLH). GABA-ergic neural systems are known to modulate activity in the nigrostriatal system and neocortex. The GABA-transaminase inhibitor, [gamma]-acetylenic GABA (GAG) was administered to rats eating in response to ESLH. This drug produced hypersynchronization of the cortical EEG and behavioral sedation, but did not alter the current threshold for evoking eating by ESLH. Implications of results for understanding mechanisms underlying ESLH-induced eating are discussed. | en_US |
dc.format.extent | 462041 bytes | |
dc.format.extent | 3118 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.language.iso | en_US | |
dc.publisher | Elsevier | en_US |
dc.title | An inability of [gamma]-acetylenic GABA to block eating evoked by hypothalamic stimulation | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Public Health | en_US |
dc.subject.hlbsecondlevel | Psychology | en_US |
dc.subject.hlbsecondlevel | Neurosciences | en_US |
dc.subject.hlbsecondlevel | Molecular, Cellular and Developmental Biology | en_US |
dc.subject.hlbtoplevel | Social Sciences | en_US |
dc.subject.hlbtoplevel | Science | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | Dept. of Psychology and Neuroscience, University of Michigan, Ann Arbor, Mich. 48109 (U.S.A.) | en_US |
dc.contributor.affiliationum | Dept. of Psychology and Neuroscience, University of Michigan, Ann Arbor, Mich. 48109 (U.S.A.) | en_US |
dc.identifier.pmid | 7295383 | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/23406/1/0000351.pdf | en_US |
dc.identifier.source | Behavioural Brain Research | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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