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Antibodies to nucleic acid antigens in selective IgA deficiency
dc.contributor.authorPetty, Ross E.en_US
dc.contributor.authorHaddow, M.en_US
dc.contributor.authorOen, K.en_US
dc.contributor.authorBees, W.en_US
dc.contributor.authorCassidy, James T.en_US
dc.contributor.authorTubergen, D. G.en_US
dc.date.accessioned2006-04-07T17:34:37Z
dc.date.available2006-04-07T17:34:37Z
dc.date.issued1979-06en_US
dc.identifier.citationPetty, R. E., Haddow, M., Oen, K., Bees, W., Cassidy, J. T., Tubergen, D. G. (1979/06)."Antibodies to nucleic acid antigens in selective IgA deficiency." Clinical Immunology and Immunopathology 13(2): 182-186. <http://hdl.handle.net/2027.42/23558>en_US
dc.identifier.urihttp://www.sciencedirect.com/science/article/B6WCK-4BJW18W-C8/2/c0a461b6c49bfae0bd170679b4d8210fen_US
dc.identifier.urihttps://hdl.handle.net/2027.42/23558
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=313302&dopt=citationen_US
dc.description.abstractAntibodies to nucleic acid antigens were measured in symptomatic and asymptomatic IgA-deficient individuals, non-IgA-deficient blood donors, and patients with systemic lupus erythematosus (SLE). There was no increase in mean levels of antibodies to nucleic acid antigens (native or denatured DNA, transfer RNA) in the IgA-deficient group, although 4 of 100 IgA-deficient blood donors had persistently increased levels of antibody to native DNA. IgA deficiency has been previously shown to be associated with SLE, but there does not appear to be an intrinsic association between IgA deficiency and antibodies to nucleic acid antigens.en_US
dc.format.extent244795 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherElsevieren_US
dc.titleAntibodies to nucleic acid antigens in selective IgA deficiencyen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelMicrobiology and Immunologyen_US
dc.subject.hlbsecondlevelInternal Medicine and Specialtiesen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Medicine, University of Michigan, Ann Arbor, Michigan 48104, USA; Department of Pediatrics, University of Michigan, Ann Arbor, Michigan 48104, USAen_US
dc.contributor.affiliationotherThe Rheumatic Diseases Unit, University of Manitoba, Winnipeg, Manitoba, Canada; Department of Pediatrics, University of Manitoba, Winnipeg, Manitoba, Canadaen_US
dc.contributor.affiliationotherThe Rheumatic Diseases Unit, University of Manitoba, Winnipeg, Manitoba, Canada; Department of Pediatrics, University of Manitoba, Winnipeg, Manitoba, Canadaen_US
dc.contributor.affiliationotherThe Rheumatic Diseases Unit, University of Manitoba, Winnipeg, Manitoba, Canada; Department of Pediatrics, University of Manitoba, Winnipeg, Manitoba, Canadaen_US
dc.contributor.affiliationotherThe Rheumatic Diseases Unit, University of Manitoba, Winnipeg, Manitoba, Canada; Department of Pediatrics, University of Manitoba, Winnipeg, Manitoba, Canadaen_US
dc.contributor.affiliationotherUniversity of Colorado, Denver, Colorado 80218, USA; Department of Oncology, Denver Children's Hospital, Denver, Colorado 80218, USAen_US
dc.identifier.pmid313302en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/23558/1/0000518.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1016/0090-1229(79)90062-Xen_US
dc.identifier.sourceClinical Immunology and Immunopathologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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