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Action spectrum of the retinal mechanism mediating nocturnal light-induced suppression of rat pineal gland N-acetyltransferase

dc.contributor.authorBronstein, David M.en_US
dc.contributor.authorJacobs, Gerald H.en_US
dc.contributor.authorHaak, Katherine A.en_US
dc.contributor.authorNeitz, Jayen_US
dc.contributor.authorLytle, Loy D.en_US
dc.date.accessioned2006-04-07T19:55:44Z
dc.date.available2006-04-07T19:55:44Z
dc.date.issued1987-03-17en_US
dc.identifier.citationBronstein, David M., Jacobs, Gerald H., Haak, Katherine A., Neitz, Jay, Lytle, Loy D. (1987/03/17)."Action spectrum of the retinal mechanism mediating nocturnal light-induced suppression of rat pineal gland N-acetyltransferase." Brain Research 406(1-2): 352-356. <http://hdl.handle.net/2027.42/26770>en_US
dc.identifier.urihttp://www.sciencedirect.com/science/article/B6SYR-4834JR8-WP/2/4299a75ee8463cde1644ca3e9c7f736ben_US
dc.identifier.urihttps://hdl.handle.net/2027.42/26770
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=3567633&dopt=citationen_US
dc.description.abstractThe spectral properties of the retinal mechanism mediating the inhibitory effects of nocturnal light on pineal gland N-acetyltransferase (NAT) activity were determined. Pineal gland NAT activity declined linearly in albino rats exposed to different irradiances of a 460 or 580 nm monochromatic light during the middle of the dark phase of the cycle. The difference in sensitivity to the test lights is that predicted for a photopigment having peak absorbance at 495 nm, suggesting the inhibition of pineal gland N-acetyltransferase activity is mediated by the photopigment found in rat rods.en_US
dc.format.extent405335 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherElsevieren_US
dc.titleAction spectrum of the retinal mechanism mediating nocturnal light-induced suppression of rat pineal gland N-acetyltransferaseen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelPublic Healthen_US
dc.subject.hlbsecondlevelNeurosciencesen_US
dc.subject.hlbsecondlevelMolecular, Cellular and Developmental Biologyen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Psychiatry, Mental Health Research Institute, The University of Michigan, Ann Arbor, MI 48109, U.S.A.en_US
dc.contributor.affiliationotherDepartment of Psychology, University of California, Santa Barbara, CA 93106, U.S.A.en_US
dc.contributor.affiliationotherDepartment of Psychology, University of California, Santa Barbara, CA 93106, U.S.A.en_US
dc.contributor.affiliationotherDepartment of Psychology, University of California, Santa Barbara, CA 93106, U.S.A.en_US
dc.contributor.affiliationotherDepartment of Psychology, University of California, Santa Barbara, CA 93106, U.S.A.en_US
dc.identifier.pmid3567633en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/26770/1/0000322.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1016/0006-8993(87)90806-7en_US
dc.identifier.sourceBrain Researchen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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