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Effects of encainide and amiodarone on the adaptation of ventricular refractoriness to an increase in rate

dc.contributor.authorRosenheck, Shimonen_US
dc.contributor.authorSchmaltz, Stephen P.en_US
dc.contributor.authorKadish, Alan H.en_US
dc.contributor.authorMorady, Freden_US
dc.date.accessioned2006-04-10T13:39:44Z
dc.date.available2006-04-10T13:39:44Z
dc.date.issued1990-07-15en_US
dc.identifier.citationRosenheck, Shimon, Schmaltz, Stephen, Kadish, Alan H., Morady, Fred (1990/07/15)."Effects of encainide and amiodarone on the adaptation of ventricular refractoriness to an increase in rate." The American Journal of Cardiology 66(2): 229-230. <http://hdl.handle.net/2027.42/28462>en_US
dc.identifier.urihttp://www.sciencedirect.com/science/article/B6T10-4C6CRDW-4V/2/c3e5098532bc96622be8f285321593a8en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/28462
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=2115289&dopt=citationen_US
dc.description.abstractThe ventricular effective refractory period (VERP) adapts gradually to an increase in rate, and up to several minutes may be necessary before the maximum shortening of VERP is realized when the rate increases.1,2 Quinidine and mexiletine have been demonstrated to have no effect on the adaptation of ventricular refractoriness to an increase in rate.3 However, the effects of class IC and class III antiarrhythmic drugs on the adaptation of refractoriness to an increase in rate are not known. Therefore, the purpose of this study was to evaluate the effects of encainide and amiodarone on the adaptation of the VERP to an increase in rate.en_US
dc.format.extent269167 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherElsevieren_US
dc.titleEffects of encainide and amiodarone on the adaptation of ventricular refractoriness to an increase in rateen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelInternal Medicine and Specialtiesen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumFrom the Division of Cardiology, B1F245, and the Clinical Research Center, Department of Internal Medicine, University of Michigan Medical Center, Ann Arbor, Michigan 48109-0022, USAen_US
dc.contributor.affiliationumFrom the Division of Cardiology, B1F245, and the Clinical Research Center, Department of Internal Medicine, University of Michigan Medical Center, Ann Arbor, Michigan 48109-0022, USAen_US
dc.contributor.affiliationumFrom the Division of Cardiology, B1F245, and the Clinical Research Center, Department of Internal Medicine, University of Michigan Medical Center, Ann Arbor, Michigan 48109-0022, USAen_US
dc.contributor.affiliationumFrom the Division of Cardiology, B1F245, and the Clinical Research Center, Department of Internal Medicine, University of Michigan Medical Center, Ann Arbor, Michigan 48109-0022, USAen_US
dc.identifier.pmid2115289en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/28462/1/0000253.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1016/0002-9149(90)90596-Sen_US
dc.identifier.sourceThe American Journal of Cardiologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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