Effect of empiric antiarrhythmic therapy in resuscitated out-of-hospital cardiac arrest victims with coronary artery disease
dc.contributor.author | Moosvi, Ali R. | en_US |
dc.contributor.author | Goldstein, Sidney | en_US |
dc.contributor.author | Medendorp, Sharon VanderBrug | en_US |
dc.contributor.author | Landis, J. Richard | en_US |
dc.contributor.author | Wolfe, Robert A. | en_US |
dc.contributor.author | Leighton, Richard | en_US |
dc.contributor.author | Ritter, George | en_US |
dc.contributor.author | Vasu, C. Mark | en_US |
dc.contributor.author | Acheson, Allyn | en_US |
dc.date.accessioned | 2006-04-10T13:44:08Z | |
dc.date.available | 2006-04-10T13:44:08Z | |
dc.date.issued | 1990-05-15 | en_US |
dc.identifier.citation | Moosvi, Ali R., Goldstein, Sidney, Medendorp, Sharon VanderBrug, Landis, J. Richard, Wolfe, Robert A., Leighton, Richard, Ritter, George, Vasu, C. Mark, Acheson, Allyn (1990/05/15)."Effect of empiric antiarrhythmic therapy in resuscitated out-of-hospital cardiac arrest victims with coronary artery disease." The American Journal of Cardiology 65(18): 1192-1197. <http://hdl.handle.net/2027.42/28573> | en_US |
dc.identifier.uri | http://www.sciencedirect.com/science/article/B6T10-4C6CR7J-5/2/a89f6db03f1b0b9b72dd2e900f6e9578 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/28573 | |
dc.identifier.uri | http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=2337028&dopt=citation | en_US |
dc.description.abstract | The effect of empiric antiarrhythmic therapy with quindine and procainamide on long-term mortality was examined in 209 patients with coronary artery disease resuscitated after out-of-hospital cardiac arrest. The antiarrhythmic agent used was determined by the patient's private physician without knowledge of the study ambulatory electrocardiogram. Of the 209 patients, procainamide was prescribed in 45 (22%), qiinidine in 48 (23%) and no antiarrhythmic therapy in 116 (55%). Digoxin therapy was initiated in 101 patients. The 2-year total survival rate for the quinidine, procainamide and nontreated patients was 61, 57 and 71% (p < 0.05), and for sudden death was 69, 69 and 89% (p < 0.01), respectively. These observations suggest that empiric antiarrhythmic therapy in survivors of out-of-hospital cardiac arrest did not affect total mortality and was associated with an increased frequency of sudden death. | en_US |
dc.format.extent | 550907 bytes | |
dc.format.extent | 3118 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.language.iso | en_US | |
dc.publisher | Elsevier | en_US |
dc.title | Effect of empiric antiarrhythmic therapy in resuscitated out-of-hospital cardiac arrest victims with coronary artery disease | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Internal Medicine and Specialties | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA; Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.contributor.affiliationum | Department of Biostatistics, University of Michigan, Ann Arbor, Michigan, USA; Henry Ford Heart and Vascular Institute, Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, USA. | en_US |
dc.identifier.pmid | 2337028 | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/28573/1/0000376.pdf | en_US |
dc.identifier.doi | http://dx.doi.org/10.1016/0002-9149(90)90972-4 | en_US |
dc.identifier.source | The American Journal of Cardiology | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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