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Conserved Linkage of Early Growth Response 4, Annexin 4, and Transforming Growth Factor [alpha] on Mouse Chromosome 6
dc.contributor.authorBarrow, Lon L.en_US
dc.contributor.authorSimin, Karlen_US
dc.contributor.authorJones, Julie M.en_US
dc.contributor.authorLee, David C.en_US
dc.contributor.authorMeisler, Miriam H.en_US
dc.date.accessioned2006-04-10T18:23:46Z
dc.date.available2006-04-10T18:23:46Z
dc.date.issued1994-01-15en_US
dc.identifier.citationBarrow, Lon L., Simin, Karl, Jones, Julie M., Lee, David C., Meisler, Miriam H. (1994/01/15)."Conserved Linkage of Early Growth Response 4, Annexin 4, and Transforming Growth Factor [alpha] on Mouse Chromosome 6." Genomics 19(2): 388-390. <http://hdl.handle.net/2027.42/31832>en_US
dc.identifier.urihttp://www.sciencedirect.com/science/article/B6WG1-45NJY5K-9H/2/a7c9c13ee80feff2344ea2f2402ee286en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/31832
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=8188273&dopt=citationen_US
dc.description.abstractThe mouse genes encoding early growth response 4 (Egr4), annexin IV (Anx4), and transforming growth factor a (Tgfa) have been mapped to a linkage group on mouse chromosome 6 that is conserved on human chromosome 2p11-p13. The genes are closely linked, with 0/215 recombinants between Anx4 and Tgfa and 1/215 recombinants between these genes and Egr4 . The genes are located approximately 2 cM distal to mnd2, a mouse mutation causing neuromuscular disease. The results demonstrate that mnd2 is located at an internal position within this conserved linkage group.en_US
dc.format.extent156528 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherElsevieren_US
dc.titleConserved Linkage of Early Growth Response 4, Annexin 4, and Transforming Growth Factor [alpha] on Mouse Chromosome 6en_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelMolecular, Cellular and Developmental Biologyen_US
dc.subject.hlbsecondlevelGeneticsen_US
dc.subject.hlbsecondlevelBiological Chemistryen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, Ann Arbor, Michigan, USA.en_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, Ann Arbor, Michigan, USA.en_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, Ann Arbor, Michigan, USA.en_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, Ann Arbor, Michigan, USA.en_US
dc.contributor.affiliationotherLineberger Comprehensive Cancer Center and Department of Microbiology and Immunology, University of North Carolina School of Medicine, Chapel Hill, North Carolina, USA.en_US
dc.identifier.pmid8188273en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/31832/1/0000779.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1006/geno.1994.1078en_US
dc.identifier.sourceGenomicsen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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