Sarcosine oxidase activity of rat liver tissue: Effect of folic acid deficiency and induced hyperthyroidism
dc.contributor.author | Swendseid, Marian E. | en_US |
dc.contributor.author | Swanson, Ann L. | en_US |
dc.contributor.author | Bethell, Frank H. | en_US |
dc.date.accessioned | 2006-04-13T15:05:20Z | |
dc.date.available | 2006-04-13T15:05:20Z | |
dc.date.issued | 1952-11 | en_US |
dc.identifier.citation | Swendseid, Marian E., Swanson, Ann L., Bethell, Frank H. (1952/11)."Sarcosine oxidase activity of rat liver tissue: Effect of folic acid deficiency and induced hyperthyroidism." Archives of Biochemistry and Biophysics 41(1): 138-142. <http://hdl.handle.net/2027.42/32539> | en_US |
dc.identifier.uri | http://www.sciencedirect.com/science/article/B6WB5-4DW3GGN-1YP/2/7ecbfc7475da9c0dfa62fdc64e82842d | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/32539 | |
dc.identifier.uri | http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=12997256&dopt=citation | en_US |
dc.description.abstract | The sarcosine oxidase content of hepatic tissue from rats made deficient in folic acid was similar to that of normal rats. This is taken as evidence that sarcosine oxidase activity is not dependent upon the presence of folic acid vitamins.The liver sarcosine content of vitamin B12-deficient rats was decreased below normal values. Since B12 supplementation did not increase the enzyme activity, it could not be ascertained whether the loss in enzyme activity was primarily due to a B12 deficiency or whether it was the result of other conditions imposed by a hyperthyroid state. | en_US |
dc.format.extent | 260651 bytes | |
dc.format.extent | 3118 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.language.iso | en_US | |
dc.publisher | Elsevier | en_US |
dc.title | Sarcosine oxidase activity of rat liver tissue: Effect of folic acid deficiency and induced hyperthyroidism | en_US |
dc.type | Article | en_US |
dc.rights.robots | IndexNoFollow | en_US |
dc.subject.hlbsecondlevel | Public Health | en_US |
dc.subject.hlbsecondlevel | Chemistry | en_US |
dc.subject.hlbsecondlevel | Chemical Engineering | en_US |
dc.subject.hlbsecondlevel | Biological Chemistry | en_US |
dc.subject.hlbtoplevel | Engineering | en_US |
dc.subject.hlbtoplevel | Science | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | The Thomas Henry Simpson Memorial Institute for Medical Research, University of Michigan, Ann Arbor, Michigan, U.S.A. | en_US |
dc.contributor.affiliationum | The Thomas Henry Simpson Memorial Institute for Medical Research, University of Michigan, Ann Arbor, Michigan, U.S.A. | en_US |
dc.contributor.affiliationum | The Thomas Henry Simpson Memorial Institute for Medical Research, University of Michigan, Ann Arbor, Michigan, U.S.A. | en_US |
dc.identifier.pmid | 12997256 | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/32539/1/0000648.pdf | en_US |
dc.identifier.doi | http://dx.doi.org/10.1016/0003-9861(52)90512-2 | en_US |
dc.identifier.source | Archives of Biochemistry and Biophysics | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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