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New insights into mechanisms of oxyradical and neutrophil mediated lung injury
dc.contributor.authorWard, Peter A.en_US
dc.contributor.authorMulligan, Michael S.en_US
dc.date.accessioned2006-09-11T17:14:32Z
dc.date.available2006-09-11T17:14:32Z
dc.date.issued1991-12en_US
dc.identifier.citationWard, P. A.; Mulligan, M. S.; (1991). "New insights into mechanisms of oxyradical and neutrophil mediated lung injury." Klinische Wochenschrift 69 (21-23): 1009-1011. <http://hdl.handle.net/2027.42/45986>en_US
dc.identifier.issn1432-1440en_US
dc.identifier.issn0023-2173en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/45986
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=1665883&dopt=citationen_US
dc.description.abstractAcute lung injury in rats developing after systemic complement activation or deposition of IgG immune complexes is complement-dependent and oxygen radical-mediated. Recent findings have shown that a soluble complement receptor (sCRl) is capable of attenuating injury. Additional studies have also demonstrated requirements for the cytokines, TNF α , and for L-arginine derived products in lung injury that follows deposition of IgG immune complexes.en_US
dc.format.extent316538 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlagen_US
dc.subject.otherComplementen_US
dc.subject.otherOxygen-radicalsen_US
dc.subject.otherMolecular Medicineen_US
dc.subject.otherBiomedicineen_US
dc.subject.otherInternal Medicineen_US
dc.subject.otherSCR1en_US
dc.subject.otherTNF αen_US
dc.subject.otherL-arginineen_US
dc.titleNew insights into mechanisms of oxyradical and neutrophil mediated lung injuryen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelMedicine (General)en_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Pathology, The University of Michigan Medical School, Ann Arboren_US
dc.contributor.affiliationumDepartment of Pathology, The University of Michigan Medical School, Ann Arboren_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid1665883en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/45986/1/109_2005_Article_BF01645148.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/BF01645148en_US
dc.identifier.sourceKlinische Wochenschriften_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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