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Self-administration of fentanyl, cocaine and ketamine: effects on the pituitary–adrenal axis in rhesus monkeys
dc.contributor.authorWoods, James H.en_US
dc.contributor.authorBroadbear, Jillian H.en_US
dc.contributor.authorWinger, Gail D.en_US
dc.date.accessioned2006-09-11T17:41:31Z
dc.date.available2006-09-11T17:41:31Z
dc.date.issued2004-11en_US
dc.identifier.citationBroadbear, Jillian H.; Winger, Gail; Woods, James H.; (2004). "Self-administration of fentanyl, cocaine and ketamine: effects on the pituitary–adrenal axis in rhesus monkeys." Psychopharmacology 176 (3-4): 398-406. <http://hdl.handle.net/2027.42/46362>en_US
dc.identifier.issn0033-3158en_US
dc.identifier.issn1432-2072en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/46362
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=15114434&dopt=citationen_US
dc.description.abstractDrugs of abuse can affect the functioning of the hypothalamic–pituitary–adrenal (HPA) axis. Acute administration of drugs that serve as reinforcers have been observed to stimulate the rat HPA axis, leading to the suggestion that these stimulatory effects may contribute to the development of drug-maintained behaviors.en_US
dc.format.extent371976 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlagen_US
dc.subject.otherAdrenocorticotropinen_US
dc.subject.otherCocaineen_US
dc.subject.otherMedicineen_US
dc.subject.otherKetamineen_US
dc.subject.otherReinforcementen_US
dc.subject.otherRhesus Monkeyen_US
dc.subject.otherBehavioren_US
dc.subject.otherFentanylen_US
dc.subject.otherSelf-administrationen_US
dc.subject.otherCortisolen_US
dc.titleSelf-administration of fentanyl, cocaine and ketamine: effects on the pituitary–adrenal axis in rhesus monkeysen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPsychiatryen_US
dc.subject.hlbsecondlevelNeurosciencesen_US
dc.subject.hlbsecondlevelChemistryen_US
dc.subject.hlbsecondlevelBiological Chemistryen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USAen_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USA; Department of Psychology, University of Michigan Medical School, Ann Arbor, MI, USAen_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USA; Department of Physiology, Monash University, Building 13F, Clayton, Victoria, 3800, Australiaen_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid15114434en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/46362/1/213_2004_Article_1891.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/s00213-004-1891-xen_US
dc.identifier.sourcePsychopharmacologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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