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The effects of CRF antagonists, antalarmin, CP154,526, LWH234, and R121919, in the forced swim test and on swim-induced increases in adrenocorticotropin in rats
dc.contributor.authorHoushyar, Hanien_US
dc.contributor.authorWoods, James H.en_US
dc.contributor.authorWood, Susan K.en_US
dc.contributor.authorRice, Kenner C.en_US
dc.contributor.authorHsin, Ling-Weien_US
dc.contributor.authorJutkiewicz, Emily M.en_US
dc.date.accessioned2006-09-11T17:41:45Z
dc.date.available2006-09-11T17:41:45Z
dc.date.issued2005-07en_US
dc.identifier.citationJutkiewicz, Emily M.; Wood, Susan K.; Houshyar, Hani; Hsin, Ling-Wei; Rice, Kenner C.; Woods, James H.; (2005). "The effects of CRF antagonists, antalarmin, CP154,526, LWH234, and R121919, in the forced swim test and on swim-induced increases in adrenocorticotropin in rats." Psychopharmacology 180(2): 215-223. <http://hdl.handle.net/2027.42/46365>en_US
dc.identifier.issn0033-3158en_US
dc.identifier.issn1432-2072en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/46365
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=15696320&dopt=citationen_US
dc.description.abstractExposure to extreme stress has been suggested to produce long-term, detrimental alterations in the hypothalamic–pituitary–adrenal (HPA) axis leading to the development of mental disorders such as depression. Therefore, compounds that block the effects of stress hormones were investigated as potential therapeutics for depression.en_US
dc.format.extent261759 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlagen_US
dc.subject.otherACTHen_US
dc.subject.otherForced Swim Testen_US
dc.subject.otherMedicineen_US
dc.subject.otherCRF Antagonistsen_US
dc.subject.otherRatsen_US
dc.titleThe effects of CRF antagonists, antalarmin, CP154,526, LWH234, and R121919, in the forced swim test and on swim-induced increases in adrenocorticotropin in ratsen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPsychiatryen_US
dc.subject.hlbsecondlevelNeurosciencesen_US
dc.subject.hlbsecondlevelChemistryen_US
dc.subject.hlbsecondlevelBiological Chemistryen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USAen_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USAen_US
dc.contributor.affiliationumDepartment of Pharmacology, University of Michigan Medical School, 1301 MSRB 3, Ann Arbor, MI, 48109-0632, USAen_US
dc.contributor.affiliationotherDepartment of Physiology, University of California San Francisco, San Francisco, CA, 94143-0444, USAen_US
dc.contributor.affiliationotherSchool of Pharmacy, College of Medicine, National Taiwan University, No.1 Jen-Ai Road, Section 1, Room 1336, Taipei, 10018, Taiwanen_US
dc.contributor.affiliationotherLaboratory of Medicinal Chemistry, NIDDK, National Institutes of Health, Bethesda, MD, USAen_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid15696320en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/46365/1/213_2005_Article_2164.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/s00213-005-2164-zen_US
dc.identifier.sourcePsychopharmacologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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