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The effects of chlorpromazine on psychomotor stimulant self-administration in the Rhesus monkey

dc.contributor.authorWilson, Marvin C.en_US
dc.contributor.authorSchuster, Charles R.en_US
dc.date.accessioned2006-09-11T17:43:02Z
dc.date.available2006-09-11T17:43:02Z
dc.date.issued1972-06en_US
dc.identifier.citationWilson, M. C.; Schuster, C. R.; (1972). "The effects of chlorpromazine on psychomotor stimulant self-administration in the Rhesus monkey." Psychopharmacologia 26(2): 115-126. <http://hdl.handle.net/2027.42/46383>en_US
dc.identifier.issn0033-3158en_US
dc.identifier.issn1432-2072en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/46383
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=4403705&dopt=citationen_US
dc.description.abstractThe effects of acute and chronic chlorpromazine treatment on psychomotor stimulant self-administration behavior in the Rhesus monkey were determined. Chlorpromazine treatment significantly increased the frequency of self-administration of cocaine, pipradrol, phenmetrazine, d -amphetamine and methylphenidate. The basis of this effect was thought to either be due to an antagonism of the reinforcing effect of these compounds or an antagonism of those actions of the psychomotor stimulants which may function in limiting their self-administration.en_US
dc.format.extent674583 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlagen_US
dc.subject.otherBiomedicineen_US
dc.subject.otherCocaineen_US
dc.subject.otherChlorpromazineen_US
dc.subject.otherPhenmetrazineen_US
dc.subject.otherPharmacology/Toxicologyen_US
dc.subject.otherSelf-Administrationen_US
dc.subject.otherMethylphenidateen_US
dc.subject.otherPsychiatryen_US
dc.titleThe effects of chlorpromazine on psychomotor stimulant self-administration in the Rhesus monkeyen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPsychiatryen_US
dc.subject.hlbsecondlevelNeurosciencesen_US
dc.subject.hlbsecondlevelChemistryen_US
dc.subject.hlbsecondlevelBiological Chemistryen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumPharmacology Department, University of Michigan, Medical School, Ann Arbor, Michigan; Department of Psychiatry, University of Chicago, Chicago, Illinoisen_US
dc.contributor.affiliationumPharmacology Department, University of Michigan, Medical School, Ann Arbor, Michigan; Department of Pharmacology, School of Pharmacy, University of Mississippi, University, Mississippien_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid4403705en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/46383/1/213_2004_Article_BF00422098.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/BF00422098en_US
dc.identifier.sourcePsychopharmacologiaen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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