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Identification of specificity H-2.7 as an erythrocyte antigen: Control by an independent locus, H-2G , between the S and D regions

dc.contributor.authorDavid, Chella S.en_US
dc.contributor.authorShreffler, Donald C.en_US
dc.contributor.authorStimpfling, Jack H.en_US
dc.date.accessioned2006-09-11T18:08:57Z
dc.date.available2006-09-11T18:08:57Z
dc.date.issued1975-12en_US
dc.identifier.citationDavid, Chella S.; Stimpfling, Jack H.; Shreffler, Donald C.; (1975). "Identification of specificity H-2.7 as an erythrocyte antigen: Control by an independent locus, H-2G , between the S and D regions." Immunogenetics 2(1): 131-139. <http://hdl.handle.net/2027.42/46742>en_US
dc.identifier.issn1432-1211en_US
dc.identifier.issn0093-7711en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/46742
dc.description.abstractSpecificity H-2.7 is expressed predominantly on erythrocytes and controlled by a gene that maps within the H-2 gene complex at a locus, designated as H-2G , which apparently lies between regions S and D . Three phenotypes have been observed with respect to this antigen: a) positive by direct test and absorption (haplotypes H-2 f , H-2 j , H-2 p , H-2 s ); b) positive only by absorption ( H-2 k ); and c) negative ( H-2 b , H-2 d , H-2 q ). New crossover positions have been established for several H-2 recombinants based on classifications for the H-2G locus.en_US
dc.format.extent445080 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlag; Springer-Verlag New York Inc.en_US
dc.subject.otherImmunologyen_US
dc.subject.otherCell Biologyen_US
dc.subject.otherBiomedicineen_US
dc.subject.otherAllergologyen_US
dc.titleIdentification of specificity H-2.7 as an erythrocyte antigen: Control by an independent locus, H-2G , between the S and D regionsen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPublic Healthen_US
dc.subject.hlbsecondlevelBiological Chemistryen_US
dc.subject.hlbtoplevelScienceen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, 48104, Ann Arbor, Michigan; McLaughlin Research Institute, Columbus Hospital, 59401, Great Falls, Montanaen_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, 48104, Ann Arbor, Michigan; McLaughlin Research Institute, Columbus Hospital, 59401, Great Falls, Montanaen_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan, 48104, Ann Arbor, Michigan; McLaughlin Research Institute, Columbus Hospital, 59401, Great Falls, Montanaen_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/46742/1/251_2005_Article_BF01572281.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/BF01572281en_US
dc.identifier.sourceImmunogeneticsen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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