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The genes for the inter-α-inhibitor family share a homologous organization in human and mouse
dc.contributor.authorDoly, Janineen_US
dc.contributor.authorSalier, Jean Philippeen_US
dc.contributor.authorVerga, Veraen_US
dc.contributor.authorDiarra-Mehropour, Maryamen_US
dc.contributor.authorErickson, Robert P.en_US
dc.date.accessioned2006-09-11T18:27:48Z
dc.date.available2006-09-11T18:27:48Z
dc.date.issued1992-12en_US
dc.identifier.citationSalier, Jean Philippe; Verga, Vera; Doly, Janine; Diarra-Mehropour, Maryam; Erickson, Robert P.; (1992). "The genes for the inter-α-inhibitor family share a homologous organization in human and mouse." Mammalian Genome 2(4): 233-239. <http://hdl.handle.net/2027.42/46989>en_US
dc.identifier.issn0938-8990en_US
dc.identifier.issn1432-1777en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/46989
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=1371941&dopt=citationen_US
dc.description.abstractInter-α-inhibitor ( IαI ) and related molecules in human are comprised of three evolutionarily related, heavy (H) chains and one light (L) chain, also termed bikunin. The latter originates from a precursor molecule that is cleaved to yield the bikunin and another protein designated α-1-microglobulin (A1m). The four H and L chains are encoded by four distinct genes designated H1, H2, H3 , and L . The L and H2 genes are localized onto human chromosomes (chr) 9 and 10, respectively, whereas the H1 and H3 genes are tandemly arranged on chr 3.en_US
dc.format.extent1328006 bytes
dc.format.extent3115 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.language.isoen_US
dc.publisherSpringer-Verlagen_US
dc.subject.otherLife Sciencesen_US
dc.subject.otherCell Biologyen_US
dc.subject.otherZoologyen_US
dc.subject.otherAnatomyen_US
dc.titleThe genes for the inter-α-inhibitor family share a homologous organization in human and mouseen_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelNatural Resources and Environmenten_US
dc.subject.hlbsecondlevelMolecular, Cellular and Developmental Biologyen_US
dc.subject.hlbsecondlevelEcology and Evolutionary Biologyen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.subject.hlbtoplevelScienceen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan Medical School, 48109-0618, Ann Arbor, Michigan, USA; Department of Pediatrics, University of Arizona Health Sciences Center, 85724, Tucson, Arizona, USAen_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan Medical School, 48109-0618, Ann Arbor, Michigan, USAen_US
dc.contributor.affiliationotherInstitut National de la Santé et de la Recherche Médicale, Unit-295, 76800, Saint-Etienne-du-Rouvray, Franceen_US
dc.contributor.affiliationotherInstitut de Recherches Scientifiques sur le Cancer, CNRS UPR-37, 94800, Villejuif, Franceen_US
dc.contributor.affiliationotherInstitut National de la Santé et de la Recherche Médicale, Unit-78, 76230, Boisguillaume, Franceen_US
dc.contributor.affiliationumcampusAnn Arboren_US
dc.identifier.pmid1371941en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/46989/1/335_2004_Article_BF00355432.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1007/BF00355432en_US
dc.identifier.sourceMammalian Genomeen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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