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dc.contributor.authorPan, Yue-Liangen_US
dc.contributor.authorKrooth, Robert S.en_US
dc.contributor.authorTakahara, Shigeoen_US
dc.date.accessioned2007-04-06T18:02:39Z
dc.date.available2007-04-06T18:02:39Z
dc.date.issued1968-04en_US
dc.identifier.citationPan, Yue-Liang; Krooth, Robert S.; Takahara, Shigeo (1968)."The influence of progressive growth on the specific catalase activity of human diploid cell strains. II. Effect of cellular genotype: A heterozygous strain." Journal of Cellular Physiology 71(2): 161-163. <http://hdl.handle.net/2027.42/49864>en_US
dc.identifier.issn0021-9541en_US
dc.identifier.issn1097-4652en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/49864
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=5674707&dopt=citationen_US
dc.description.abstractHuman diploid cell strains develop progressively higher levels of specific catalase activity as they grow. Following subculture activity falls again. A diploid cell strain heterozygous for the gene for acatalasia I (acatalasemia) was found to develop specific catalase activity at proportionately the same rate as normal cell strains. Yet the mutant gene reduced the absolute level of specific catalase activity which the culture attained at any given point in time. In this respect the heterozygous acatalasia I strain resembles the homozygous acatalasia II strain previously reported.en_US
dc.format.extent186461 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherWiley Subscription Services, Inc., A Wiley Companyen_US
dc.subject.otherLife and Medical Sciencesen_US
dc.subject.otherCell & Developmental Biologyen_US
dc.titleThe influence of progressive growth on the specific catalase activity of human diploid cell strains. II. Effect of cellular genotype: A heterozygous strainen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelMolecular, Cellular and Developmental Biologyen_US
dc.subject.hlbsecondlevelKinesiology and Sportsen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.subject.hlbtoplevelScienceen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumLawrence D. Buhl Center for Human Genetics, Department of Human Genetics, University of Michigan Medical School, Ann Arbor, Michigan ; Department of Oto-Rhino-Laryngology, Okayama University Medical School, Okayama, Japanen_US
dc.contributor.affiliationumLawrence D. Buhl Center for Human Genetics, Department of Human Genetics, University of Michigan Medical School, Ann Arbor, Michigan ; Department of Oto-Rhino-Laryngology, Okayama University Medical School, Okayama, Japanen_US
dc.contributor.affiliationumLawrence D. Buhl Center for Human Genetics, Department of Human Genetics, University of Michigan Medical School, Ann Arbor, Michigan ; Department of Oto-Rhino-Laryngology, Okayama University Medical School, Okayama, Japanen_US
dc.identifier.pmid5674707en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/49864/1/1040710206_ftp.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1002/jcp.1040710206en_US
dc.identifier.sourceJournal of Cellular Physiologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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