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The EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophy

dc.contributor.authorNewman, S. E.en_US
dc.date.accessioned2007-04-06T18:48:35Z
dc.date.available2007-04-06T18:48:35Z
dc.date.issued1978-04en_US
dc.identifier.citationNewman, S. E. (1978)."The EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophy." Annals of Neurology 3(4): 299-304. <http://hdl.handle.net/2027.42/50293>en_US
dc.identifier.issn0364-5134en_US
dc.identifier.issn1531-8249en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/50293
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=666269&dopt=citationen_US
dc.description.abstractEleven chronic alcoholic patients without other nonneurological or traumatic disease were evaluated by a simultaneous electroencephalogram and computerized axial tomogram. The findings suggested that chronic abusers of approximately 60 years of age or less may have a normal EEG despite the presence of cerebral cortical atrophy or dementia. In alcoholics over 60 years of age, the greater the severity of cerebral cortical atrophy, the greater the slowing in background frequency of the EEG. Voltage diminution and slow-wave transients also occurred more frequently in the older patients. The incidence of EEG abnormalities was greater than the incidence of CAT scan evidence for cerebral cortical atrophy in alcoholics over 60 years of age with dementia.en_US
dc.format.extent654802 bytes
dc.format.extent3118 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherWiley Subscription Services, Inc., A Wiley Companyen_US
dc.subject.otherLife and Medical Sciencesen_US
dc.subject.otherNeuroscience, Neurology, and Psychiatryen_US
dc.titleThe EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophyen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelPsychiatryen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumEEG Laboratory, Neuropsychiatric Institute, University of Michigan Medical Center, Ann Arbor, MI ; Clinical Neurosciences Branch, National Institute of Neurological and Communicative Disorders and Stroke, National Institutes of Health, Bethesda, MD 20014en_US
dc.identifier.pmid666269en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/50293/1/410030404_ftp.pdfen_US
dc.identifier.doihttp://dx.doi.org/10.1002/ana.410030404en_US
dc.identifier.sourceAnnals of Neurologyen_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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