The EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophy

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dc.contributor.author Newman, S. E. en_US
dc.date.accessioned 2007-04-06T18:48:35Z
dc.date.available 2007-04-06T18:48:35Z
dc.date.issued 1978-04 en_US
dc.identifier.citation Newman, S. E. (1978)."The EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophy." Annals of Neurology 3(4): 299-304. <http://hdl.handle.net/2027.42/50293> en_US
dc.identifier.issn 0364-5134 en_US
dc.identifier.issn 1531-8249 en_US
dc.identifier.uri http://hdl.handle.net/2027.42/50293
dc.identifier.uri http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=666269&dopt=citation en_US
dc.description.abstract Eleven chronic alcoholic patients without other nonneurological or traumatic disease were evaluated by a simultaneous electroencephalogram and computerized axial tomogram. The findings suggested that chronic abusers of approximately 60 years of age or less may have a normal EEG despite the presence of cerebral cortical atrophy or dementia. In alcoholics over 60 years of age, the greater the severity of cerebral cortical atrophy, the greater the slowing in background frequency of the EEG. Voltage diminution and slow-wave transients also occurred more frequently in the older patients. The incidence of EEG abnormalities was greater than the incidence of CAT scan evidence for cerebral cortical atrophy in alcoholics over 60 years of age with dementia. en_US
dc.format.extent 654802 bytes
dc.format.extent 3118 bytes
dc.format.mimetype application/pdf
dc.format.mimetype text/plain
dc.publisher Wiley Subscription Services, Inc., A Wiley Company en_US
dc.subject.other Life and Medical Sciences en_US
dc.subject.other Neuroscience, Neurology, and Psychiatry en_US
dc.title The EEG manifestations of chronic ethanol abuse: Relation to cerebral cortical atrophy en_US
dc.type Article en_US
dc.rights.robots IndexNoFollow en_US
dc.subject.hlbsecondlevel Psychiatry en_US
dc.subject.hlbtoplevel Health Sciences en_US
dc.description.peerreviewed Peer Reviewed en_US
dc.contributor.affiliationum EEG Laboratory, Neuropsychiatric Institute, University of Michigan Medical Center, Ann Arbor, MI ; Clinical Neurosciences Branch, National Institute of Neurological and Communicative Disorders and Stroke, National Institutes of Health, Bethesda, MD 20014 en_US
dc.identifier.pmid 666269 en_US
dc.description.bitstreamurl http://deepblue.lib.umich.edu/bitstream/2027.42/50293/1/410030404_ftp.pdf en_US
dc.identifier.doi http://dx.doi.org/10.1002/ana.410030404 en_US
dc.identifier.source Annals of Neurology en_US
dc.owningcollname Interdisciplinary and Peer-Reviewed
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