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Paraoxonase-1 Q192R polymorphism and risk of sporadic amyotrophic lateral sclerosis
dc.date.accessioned2010-04-01T15:15:50Z
dc.date.available2010-04-01T15:15:50Z
dc.date.issued2006-04en_US
dc.identifier.citation(2006). "Paraoxonase-1 Q192R polymorphism and risk of sporadic amyotrophic lateral sclerosis." Clinical Genetics 69(4): 358-359. <http://hdl.handle.net/2027.42/65735>en_US
dc.identifier.issn0009-9163en_US
dc.identifier.issn1399-0004en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/65735
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=16630171&dopt=citationen_US
dc.format.extent75958 bytes
dc.format.extent3110 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherBlackwell Publishing Ltd/Inc.en_US
dc.rightsBlackwell Munksgaard, 2006en_US
dc.titleParaoxonase-1 Q192R polymorphism and risk of sporadic amyotrophic lateral sclerosisen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelGeneticsen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDept. Neurol., University of Michigan Ann Arbor, MIen_US
dc.contributor.affiliationother* Agnieszka Slowik, MD Department of Neurology Jagiellonian University 31-503 Krakow Botaniczna 3 Poland Tel.: +48 12 424 8609 Fax: +48 12 424 8626 e-mail: slowik@neuro.cm-uj.krakow.plen_US
dc.contributor.affiliationotherDept. Neurol., Jagiellonian University Krakow, Polanden_US
dc.identifier.pmid16630171en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/65735/1/j.1399-0004.2006.00590.x.pdf
dc.identifier.doi10.1111/j.1399-0004.2006.00590.xen_US
dc.identifier.sourceClinical Geneticsen_US
dc.identifier.citedreferenceLi HL, Liu DP, Liang CC. Paraoxonase gene polymorphisms, oxidative stress, and diseases. J Mol Med 2003 : 81 : 766 – 779.en_US
dc.identifier.citedreferenceBonnefont-Rousselot D, Lacomblez L, Jaudon M et al. Blood oxidative stress in amyotrophic lateral sclerosis. J Neurol Sci 2000 : 178 : 57 – 62.en_US
dc.identifier.citedreferenceArmon C. An evidence-based medicine approach to the evaluation of the role of exogenous risk factors in sporadic amyotrophic lateral sclerosis. Neuroepidemiology 2003 : 22 : 217 – 228.en_US
dc.identifier.citedreferenceMiller RG, Munsat TL, Swash M, Brooks BR. Consensus guidelines for the design and implementation of clinical trials in ALS. World Federation of Neurology committee on Research. J Neurol Sci 1999 : 169 : 2 – 12.en_US
dc.identifier.citedreferenceOmbres D, Pannitteri G, Montali A et al. The Gln-Arg192 polymorphism of human paraoxonase gene is not associated with coronary artery disease in Italian patients. Arterioscler Thromb Vasc Biol 1998 : 18 : 1611 – 1616.en_US
dc.identifier.citedreferenceHaley RW. Excess incidence of ALS in young Gulf War veterans. Neurology 2003 : 61 : 750 – 756.en_US
dc.identifier.citedreferenceHaley R, Billecke S, La Du BN. Association of low PON1 type Q (type A) arylsterase activity with neurologic symptom complexes in Gulf War veterans. Toxicol Appl Pharmacol 1999 : 157 : 227 – 233.en_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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