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Smoking and reverse cholesterol transport: evidence for gene-environment interaction

dc.contributor.authorKaprio, Jaakkoen_US
dc.contributor.authorFerrell, Robert E.en_US
dc.contributor.authorKottke, Bruce A.en_US
dc.contributor.authorSing, Charles F.en_US
dc.date.accessioned2010-04-01T15:35:27Z
dc.date.available2010-04-01T15:35:27Z
dc.date.issued1989-10en_US
dc.identifier.citationKaprio, Jaakko; Ferrell, Robert E.; Kottke, Bruce A.; Sing, Charles F. (1989). "Smoking and reverse cholesterol transport: evidence for gene-environment interaction." Clinical Genetics 36(4): 266-268. <http://hdl.handle.net/2027.42/66077>en_US
dc.identifier.issn0009-9163en_US
dc.identifier.issn1399-0004en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/66077
dc.identifier.urihttp://www.ncbi.nlm.nih.gov/sites/entrez?cmd=retrieve&db=pubmed&list_uids=2509115&dopt=citationen_US
dc.format.extent170130 bytes
dc.format.extent3110 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherBlackwell Publishing Ltden_US
dc.rights1989 Blackwell Munksgaarden_US
dc.titleSmoking and reverse cholesterol transport: evidence for gene-environment interactionen_US
dc.typeArticleen_US
dc.rights.robotsIndexNoFollowen_US
dc.subject.hlbsecondlevelGeneticsen_US
dc.subject.hlbtoplevelHealth Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumDepartment of Human Genetics, University of Michigan Medical School, Ann Arbor, Michiganen_US
dc.contributor.affiliationotherDivision of Cardiovascular Diseases, Mayo Clinic and Mayo Foundation, Rochester, Minnesotaen_US
dc.contributor.affiliationotherHuman Genetics Division, Graduate School of Public Health, University of Pittsburgh, Pittsburgh, Pennsylvania, USAen_US
dc.identifier.pmid2509115en_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/66077/1/j.1399-0004.1989.tb03201.x.pdf
dc.identifier.doi10.1111/j.1399-0004.1989.tb03201.xen_US
dc.identifier.sourceClinical Geneticsen_US
dc.identifier.citedreferenceBerg, K., A.-L. Berresen & G. Dahlen ( 1979 ). Effect of smoking on serum levels of HDL apoproteins. Atherosclerosis 34, 339 – 343.en_US
dc.identifier.citedreferenceKamboh, M. I., R. E. Ferrell & B. Sepehrnia ( 1988 ). Genetic studies of human apolipoproteins. IV. Structural heterogeneity of apolipoprotein H (beta-2-glycoprotein I). Am. J. Hum. Genet. 42, 452 – 457.en_US
dc.identifier.citedreferenceKondo, I., K. Berg, D. Drayna & R. Lawn ( 1989 ) DNA polymorphism at the locus for human cholesteryl ester transfer protein (CETP) is associated with high density lipoprotein cholesterol and apolipoprotein levels. Clin. Genet. 35, 49 – 56.en_US
dc.identifier.citedreferenceKaprio, J., R. E. Ferrell, B. A. Kottke, S. T. Turner & C. F. Sing ( 1989 ). Effect of polymorphisms in apolipoprotein E, A-IV and H on quantitative traits related to risk for cardiovascular disease. In preparation.en_US
dc.identifier.citedreferenceLaustiola, K., R. Lassila, J. Kaprio & M. Koskenvuo ( 1988 ). Decreased Β-adrenergic receptor density and catecholamine response in male cigarette smokers. A study of monozygotic twin pairs discordant for smoking. Circulation 78, 1234 – 1240.en_US
dc.identifier.citedreferenceMoll, P., V. Michels, W. Weidman & B. Kottke ( 1989 ). The genetic determination of plasma apolipoprotein AI in a population-based sample. Am. J. Hum. Genet. 44, 124 – 139.en_US
dc.identifier.citedreferenceSAS Institute Inc. ( 1985 ). SAS User's Guide: Statistics, Version 5 Edit. Cary, NC : Sas Institute Inc..en_US
dc.identifier.citedreferenceTurner, S. T., W. Weidman, V. Michels, T. Reed, C. Ormson, T. Fuller & C. F. Sing ( 1989 ). Distribution of sodium-lithium countertransport and blood pressure in Caucasians 5 to 89 years of age. Hypertension 13, 378 – 391.en_US
dc.identifier.citedreferenceWilhelmseni L., K. Svardsudd, K. Korsen-Bengtsen, B. Larsson, L. Welin & G. Tibblin ( 1984 ). Fibrinogen as a risk factor for stroke and myocardial infarction. N. Engl. J. Med. 311, 501 – 505.en_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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