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Drugs, Environment, Brain, and Behavior
dc.contributor.authorCrombag, Hans S.en_US
dc.contributor.authorRobinson, Terry E.en_US
dc.date.accessioned2010-06-01T19:58:54Z
dc.date.available2010-06-01T19:58:54Z
dc.date.issued2004-06en_US
dc.identifier.citationCrombag, Hans S.; Robinson, Terry E. (2004). "Drugs, Environment, Brain, and Behavior." Current Directions in Psychological Science 13(3): 107-111. <http://hdl.handle.net/2027.42/73110>en_US
dc.identifier.issn0963-7214en_US
dc.identifier.issn1467-8721en_US
dc.identifier.urihttps://hdl.handle.net/2027.42/73110
dc.description.abstractThe effects of psychoactive drugs are not just a function of their pharmacological actions, but are due to complex interactions among pharmacological, psychological, and environmental factors. We discuss here how drug-environment interactions determine the likelihood that addictive drugs produce a persistent form of neurobehavioral plasticity (sensitization) thought to be involved in the pathophysiology of addiction and relapse.en_US
dc.format.extent216207 bytes
dc.format.extent3109 bytes
dc.format.mimetypeapplication/pdf
dc.format.mimetypetext/plain
dc.publisherBlackwell Publishingen_US
dc.rightsCopyright © 2004 American Psychological Societyen_US
dc.subject.otherSensitizationen_US
dc.subject.otherNeuroplasticityen_US
dc.subject.otherAddictionen_US
dc.subject.otherRelapseen_US
dc.subject.otherGenesen_US
dc.titleDrugs, Environment, Brain, and Behavioren_US
dc.typeArticleen_US
dc.subject.hlbsecondlevelPsychologyen_US
dc.subject.hlbtoplevelSocial Sciencesen_US
dc.description.peerreviewedPeer Revieweden_US
dc.contributor.affiliationumUniversity of Michigan, Ann Arboren_US
dc.description.bitstreamurlhttp://deepblue.lib.umich.edu/bitstream/2027.42/73110/1/j.0963-7214.2004.00285.x.pdf
dc.identifier.doi10.1111/j.0963-7214.2004.00285.xen_US
dc.identifier.sourceCurrent Directions in Psychological Scienceen_US
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dc.identifier.citedreferenceBadiani, A., Oates, M. M., Day, H. E., Watson, S. J., Akil, H., & Robinson, T. E. ( 1998 ). Amphetamine-induced behavior, dopamine release, and c-fos mRNA expression: Modulation by environmental novelty. The Journal of Neuroscience, 18, 10579 – 10593.en_US
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dc.identifier.citedreferenceCrombag, H. S., Badiani, A., Chan, J., Dell'Orco, J., Dineen, S. P., & Robinson, T. E. ( 2001 ). The ability of environmental context to facilitate psychomotor sensitization to amphetamine can be dissociated from its effect on acute drug responsiveness and on conditioned responding. Neuropsychopharmacology, 24, 680 – 690.en_US
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dc.identifier.citedreferenceCrombag, H. S., Grimm, J. W., & Shaham, Y. ( 2002 ). Effect of dopamine receptor antagonists on renewal of cocaine seeking by reexposure to drug-associated contextual cues. Neuropsychopharmacology, 27, 1006 – 1015.en_US
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dc.identifier.citedreferencePaolone, G., Burdino, R., & Badiani, A. ( 2003 ). Dissociation in the modulatory effects of environmental novelty on the locomotor, analgesic, and eating response to acute and repeated morphine in the rat. Psychopharmacology, 166 ( 2 ), 146 – 155.en_US
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dc.identifier.citedreferenceUslaner, J., Badiani, A., Day, H. E., Watson, S. J., Akil, H., & Robinson, T. E. ( 2001 ). Environmental context modulates the ability of cocaine and amphetamine to induce c-fos mRNA expression in the neocortex, caudate nucleus, and nucleus accumbens. Brain Research, 920 ( 1–2 ), 106 – 116.en_US
dc.owningcollnameInterdisciplinary and Peer-Reviewed


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