Protein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Association
dc.contributor.author | Nistor, Ioana | en_US |
dc.contributor.author | Nair, Rajan P. | en_US |
dc.contributor.author | Stuart, Philip | en_US |
dc.contributor.author | Hiremagalore, Ravi | en_US |
dc.contributor.author | Thompson, Rachel A. | en_US |
dc.contributor.author | Jenisch, Stefan | en_US |
dc.contributor.author | Weichenthal, Michael | en_US |
dc.contributor.author | Abecasis, Gonçalo R. | en_US |
dc.contributor.author | Qin, Zhaohui Steve | en_US |
dc.contributor.author | Christophers, Enno | en_US |
dc.contributor.author | Lim, Henry W. | en_US |
dc.contributor.author | Voorhees, John J. | en_US |
dc.contributor.author | Elder, James T. | en_US |
dc.date.accessioned | 2010-06-01T21:21:43Z | |
dc.date.available | 2010-06-01T21:21:43Z | |
dc.date.issued | 2005-05 | en_US |
dc.identifier.citation | Nistor, Ioana; Nair, Rajan P.; Stuart, Philip; Hiremagalore, Ravi; Thompson, Rachel A.; Jenisch, Stefan; Weichenthal, Michael; Abecasis, GonÇalo R.; Qin, Zhaohui S.; Christophers, Enno; Lim, Henry W.; Voorhees, John J.; Elder, James T. (2005). "Protein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Association." Journal of General Internal Medicine 20(5): 395-396. <http://hdl.handle.net/2027.42/74426> | en_US |
dc.identifier.issn | 0884-8734 | en_US |
dc.identifier.issn | 1525-1497 | en_US |
dc.identifier.uri | https://hdl.handle.net/2027.42/74426 | |
dc.format.extent | 57066 bytes | |
dc.format.extent | 3109 bytes | |
dc.format.mimetype | application/pdf | |
dc.format.mimetype | text/plain | |
dc.publisher | Blackwell Science Inc | en_US |
dc.rights | Copyright © 2005 by The Society for Investigative Dermatology, Inc. | en_US |
dc.title | Protein Tyrosine Phosphatase Gene PTPN22 Polymorphism in Psoriasis: Lack of Evidence for Association | en_US |
dc.type | Article | en_US |
dc.subject.hlbsecondlevel | Internal Medicine and Specialties | en_US |
dc.subject.hlbtoplevel | Health Sciences | en_US |
dc.description.peerreviewed | Peer Reviewed | en_US |
dc.contributor.affiliationum | Department of Dermatology, University of Michigan Medical School, Ann Arbor, Michigan, USA ; Departments of | en_US |
dc.contributor.affiliationum | § Department of Biostatistics, Center for Statistical Genetics, School of Public Health, University of Michigan, Ann Arbor, Michigan, USA ; | en_US |
dc.contributor.affiliationum | ¶ Department of Dermatology, Henry Ford Hospital, Detroit, Michigan, USA ; | en_US |
dc.contributor.affiliationum | ∥ Department of Radiation Oncology (Cancer Biology), University of Michigan Medical School, Ann Arbor, Michigan, USA ; | en_US |
dc.contributor.affiliationum | # Ann Arbor Veterans Affairs Hospital, Ann Arbor, Michigan, USA | en_US |
dc.contributor.affiliationother | † Immunology and | en_US |
dc.contributor.affiliationother | † Dermatology, University of Kiel, Kiel, Germany ; | en_US |
dc.description.bitstreamurl | http://deepblue.lib.umich.edu/bitstream/2027.42/74426/1/j.0022-202X.2005.23802.x.pdf | |
dc.identifier.doi | 10.1111/j.0022-202X.2005.23802.x | en_US |
dc.identifier.source | Journal of General Internal Medicine | en_US |
dc.identifier.citedreference | Begovich AB, Caillier SJ, Alexander HC, Penko JM, Hauser SL, Barcellos LF, Oksenberg JR: The R620W polymorphism of the protein tyrosine phosphatase PTPN22 is not associated with multiple sclerosis. Am J Hum Genet 76: 184 – 187, 2005 | en_US |
dc.identifier.citedreference | Begovich AB, Carlton VE, Honigberg LA, Schrodi SJ, Chokkalingam AP, Alexander HC, Ardlie KG, et al: A missense single-nucleotide polymorphism in a gene encoding a protein tyrosine phosphatase (PTPN22) is associated with rheumatoid arthritis. Am J Hum Genet 75: 330 – 337, 2004 | en_US |
dc.identifier.citedreference | Bottini N, Musumeci L, Alonso A, et al: A functional variant of lymphoid tyrosine phosphatase is associated with type I diabetes. Nat Genet 36: 337 – 338, 2004 | en_US |
dc.identifier.citedreference | Cloutier JF, Veillette A: Association of inhibitory tyrosine protein kinase p50csk with protein tyrosine phosphatase PEP in T cells and other hemopoietic cells. EMBO J 15: 4909 – 4918, 1996 | en_US |
dc.identifier.citedreference | Cloutier JF, Veillette A: Cooperative inhibition of T-cell antigen receptor signaling by a complex between a kinase and a phosphatase. J Exp Med 189: 111 – 121, 1999 | en_US |
dc.identifier.citedreference | Collaboration: Fine mapping of the psoriasis susceptibiliity gene PSORS1: A reassessment of risk associated with a putative risk haplotype lacking HLA-Cw6. J Invest Dermatol, 2005, in press | en_US |
dc.identifier.citedreference | Criswell LA, Pfeiffer KA, Lum RF, et al: Analysis of families in the Multiple Autoimmune Disease Genetics Consortium (MADGC) collection: The PTPN22 620W allele associates with multiple autoimmune phenotypes. Am J Hum Genet 76: 561 – 571, 2005 | en_US |
dc.identifier.citedreference | Horvath S, Xu X, Laird NM: The family based association test method: Strategies for studying general genotype–phenotype associations. Eur J Hum Genet 9: 301 – 306, 2001 | en_US |
dc.identifier.citedreference | Horvath S, Xu X, Lake SL, Silverman EK, Weiss ST, Laird NM: Family-based tests for associating haplotypes with general phenotype data: Application to asthma genetics. Genet Epidemiol 26: 61 – 69, 2004 | en_US |
dc.identifier.citedreference | Knapp M: A note on power approximations for the transmission/disequilibrium test. Am J Hum Genet 64: 1177 – 1185, 1999 | en_US |
dc.identifier.citedreference | Kyogoku C, Langefeld CD, Ortmann WA, et al: Genetic association of the R620W polymorphism of protein tyrosine phosphatase PTPN22 with human SLE. Am J Hum Genet 75: 504 – 507, 2004 | en_US |
dc.identifier.citedreference | Ladner MB, Bottini N, Valdes AM, Noble JA: Association of the single nucleotide polymorphism C1858T of the PTPN22 gene with type 1 diabetes. Hum Immunol 66: 60 – 64, 2005 | en_US |
dc.identifier.citedreference | Lew W, Bowcock AM, Krueger JG: Psoriasis vulgaris: Cutaneous lymphoid tissue supports T-cell activation and “Type 1” inflammatory gene expression. Trends Immunol 25: 295 – 305, 2004 | en_US |
dc.identifier.citedreference | Martin ER, Bass MP, Kaplan NL: Correcting for a potential bias in the pedigree disequilibrium test. Am J Hum Genet 68: 1065 – 1067, 2001 | en_US |
dc.identifier.citedreference | Martin ER, Monks SA, Warren LL, Kaplan NL: A test for linkage and association in general pedigrees: The pedigree disequilibrium test. Am J Hum Genet 67: 146 – 154, 2000 | en_US |
dc.identifier.citedreference | Onengut-Gumuscu S, Ewens KG, Spielman RS, Concannon P: A functional polymorphism (1858C/T) in the PTPN22 gene is linked and associated with type I diabetes in multiplex families. Genes Immun 5: 678 – 680, 2004 | en_US |
dc.identifier.citedreference | Rabinowitz D, Laird N: A unified approach to adjusting association tests for population admixture with arbitrary pedigree structure and arbitrary missing marker information. Hum Heredity 50: 211 – 223, 2000 | en_US |
dc.identifier.citedreference | Smyth D, Cooper JD, Collins JE, et al: Replication of an association between the lymphoid tyrosine phosphatase locus (LYP/PTPN22) with type 1 diabetes, and evidence for its role as a general autoimmunity locus. Diabetes 53: 3020 – 3023, 2004 | en_US |
dc.identifier.citedreference | Spielman RS, Ewens WJ: The TDT and other family-based tests for linkage disequilibrium and association. Am J Hum Genet 59: 983 – 989, 1996 | en_US |
dc.identifier.citedreference | Spielman RS, McGinnis RE, Ewens WJ: Transmission test for linkage disequilibrium: The insulin gene region and insulin-dependent diabetes mellitus (IDDM). Am J Hum Genet 52: 506 – 516, 1993 | en_US |
dc.identifier.citedreference | Sugiyama H, Gyulai R, Toichi E, et al: Dysfunctional blood and target tissue CD4+CD25 high regulatory T cells in psoriasis: Mechanism underlying unrestrained pathogenic effector T cell proliferation. J Immunol 174: 164 – 173, 2005 | en_US |
dc.identifier.citedreference | Velaga MR, Wilson V, Jennings CE, et al: The codon 620 tryptophan allele of the lymphoid tyrosine phosphatase (LYP) gene is a major determinant of Graves' disease. J Clin Endocrinol Metab 89: 5862 – 5865, 2004 | en_US |
dc.owningcollname | Interdisciplinary and Peer-Reviewed |
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